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Karin H. Humphries

Researcher at University of British Columbia

Publications -  237
Citations -  15057

Karin H. Humphries is an academic researcher from University of British Columbia. The author has contributed to research in topics: Population & Myocardial infarction. The author has an hindex of 58, co-authored 221 publications receiving 13287 citations. Previous affiliations of Karin H. Humphries include Provincial Health Services Authority & Erasmus University Rotterdam.

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Percutaneous transarterial aortic valve replacement in selected high-risk patients with aortic stenosis.

TL;DR: Percutaneous valve replacement may be an alternative to conventional open heart surgery in selected high-risk patients with severe symptomatic aortic stenosis in whom there was a consensus that the risks of conventional openheart surgery were very high.
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Spontaneous Coronary Artery Dissection Association With Predisposing Arteriopathies and Precipitating Stressors and Cardiovascular Outcomes

TL;DR: Nonatherosclerotic spontaneous coronary artery dissection (NA-SCAD) is underdiagnosed and an important cause of myocardial infarction in young women and survivors are at risk for recurrent cardiovascular events, including recurrent SCAD.
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Transcatheter aortic valve implantation: impact on clinical and valve-related outcomes.

TL;DR: Consecutive high-risk patients who had been declined as surgical candidates but who underwent successful transcatheter aortic valve implantation with a balloon-expandable valve between January 2005 and December 2006 and survived past 30 days were assessed.
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Visceral adipose tissue accumulation differs according to ethnic background: results of the Multicultural Community Health Assessment Trial (M-CHAT)

TL;DR: Compared with Europeans, the Chinese and South Asian cohorts had a relatively greater amount of abdominal adipose tissue, and this difference was more pronounced with VAT.
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A comprehensive view of sex-specific issues related to cardiovascular disease

TL;DR: The lack of good trial evidence concerning sex-specific outcomes has led to assumptions about CVD treatment in women, which may have resulted in inadequate diagnoses and suboptimal management, greatly affecting outcomes, and may also explain why cardiovascular health in women is not improving as fast as that of men.