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Katsuji Ikekubo

Researcher at University of California, Davis

Publications -  89
Citations -  2067

Katsuji Ikekubo is an academic researcher from University of California, Davis. The author has contributed to research in topics: Thyroid & Asialoglycoprotein receptor. The author has an hindex of 23, co-authored 89 publications receiving 2038 citations.

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Detection and properties of TSH-binding inhibitor immunoglobulins in patients with Graves' disease and Hashimoto's thyroiditis.

TL;DR: TSH-binding inhibitor immunoglobulins (TBII) have been detected in patients with Graves' disease and Hashimoto's thyroiditis by using the radioreceptor assay of TSH, and in untreated Graves' patients, TBII levels correlated well with thyroidal 99mTc uptake at 30 min and the grade of epithelial hyperplasia of thyroid follicles.
Journal Article

Regional metabolic abnormality in relation to perfusion and wall motion in patients with myocardial infarction: assessment with emission tomography using an iodinated branched fatty acid analog.

TL;DR: BMIPP imaging may play a major role in increasing the understanding of the relationship between perfusion and wall motion, particularly in patients with acute myocardial infarction and those who received revascularization therapy.
Journal Article

A New Imaging Agent for Medullary Carcinoma of the Thyroid

TL;DR: Significant uptake of the tracer was found in the clinically palpable cervical tumor masses, metastatic sites, and residual tumor in patients with pathologically confirmed medullary thyroid carcinoma and elevated serum calcitonin values.
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A Novel Homozygous Missense Mutation of Melanocortin-4 Receptor (MC4R) in a Japanese Woman With Severe Obesity

TL;DR: A novel homozygous missense mutation of MC4R (G98R) is reported in a nondiabetic Japanese woman with severe early-onset obesity, which is located in its second transmembrane domain, indicating a severe loss-of-function mutation.
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Correlation of the antibody titers with serum prolactin levels and their clinical course in patients with anti-prolactin autoantibody

TL;DR: Most patients with anti-PRL autoantibody lacked the clinical symptoms of hyperprolactinemia, such as amenorrhea and galactorrhea, and spontaneous pregnancy occurred despite the marked hyper Prolactinemic state, indicating that the biological activity of PRL was attenuated by the autoantIBody.