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Kleopatra H. Schulpis

Researcher at Boston Children's Hospital

Publications -  167
Citations -  2731

Kleopatra H. Schulpis is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Na+/K+-ATPase & Galactosemia. The author has an hindex of 27, co-authored 166 publications receiving 2516 citations. Previous affiliations of Kleopatra H. Schulpis include Athens State University & National and Kapodistrian University of Athens.

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Psychological and sympatho-adrenal status in patients with cystic acne

TL;DR: Patients with cystic acne frequently report feeling self conscious, anxiety and social isolation, according to a national survey of patients with the condition.
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In vivo effects of high phenylalanine blood levels on Na+,K+-ATPase, Mg2+-ATPase activities and biogenic amine concentrations in phenylketonuria.

TL;DR: High Phe and/or low NA, DA, 5HT plasma levels may indirectly inhibit the erythrocyte membrane Na+,K+-ATPase and Mg2+- ATPase in PKU patients, and the observed enzyme inhibitions could be a very informative peripheral marker as regards the neurotoxic Phe brain effects.
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Maternal – neonatal serum selenium and copper levels in Greeks and Albanians

TL;DR: The low Se and Cu levels evaluated in the Albanian mothers and their newborns could be related to their poor animal protein intake which could be the consequence of their low socioeconomic status.
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Morning preprandial plasma ghrelin and catecholamine concentrations in patients with phenylketonuria and normal controls: evidence for catecholamine-mediated ghrelin regulation.

TL;DR: The absence of a correlation between ghrelin and catecholamines, energy intake, or BMI in PKU patients on an inadequate diet may be due to dysregulation of their neuroendocrine system and might be affected by high Phe levels in the stomach and/or central nervous system.
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Serum Carnitine Level in Phenylketonuric Children under Dietary Control in Greece

TL;DR: Although total, free and esterified carnitine blood levels were found to be low (p < 0.001) in phenylketonuric patients under dietary treatment compared to controls, no clinical signs of Carnival deficiency were noticed.