L
László Rosivall
Researcher at Semmelweis University
Publications - 169
Citations - 5181
László Rosivall is an academic researcher from Semmelweis University. The author has contributed to research in topics: Kidney disease & Renin–angiotensin system. The author has an hindex of 38, co-authored 162 publications receiving 4607 citations. Previous affiliations of László Rosivall include Hungarian Academy of Sciences & Education and Research Network.
Papers
More filters
Journal ArticleDOI
Elevated Fibroblast Growth Factor 23 is a Risk Factor for Kidney Transplant Loss and Mortality
Myles Wolf,Miklos Z. Molnar,Miklos Z. Molnar,Ansel P. Amaral,Maria E. Czira,Anna Rudas,Akos Ujszaszi,István Kiss,László Rosivall,János P. Kósa,Peter L. Lakatos,Csaba P. Kovesdy,Istvan Mucsi,Istvan Mucsi +13 more
TL;DR: It is concluded that a high (or elevated) FGF23 is an independent risk factor for death and allograft loss in kidney transplant recipients, and other measures of phosphorus metabolism did not consistently associate with outcomes.
Journal ArticleDOI
Central role for Rho in TGF-β1-induced α-smooth muscle actin expression during epithelial-mesenchymal transition
András Masszi,András Masszi,Caterina Di Ciano,Gábor Sirokmány,William T. Arthur,Ori D. Rotstein,Jiaxu Wang,Christopher A. McCulloch,László Rosivall,Istvan Mucsi,Istvan Mucsi,Andras Kapus +11 more
TL;DR: New research suggests that, during tubulointerstitial fibrosis, α-smooth muscle actin (SMA)-expressing mesenchymal cells might derive from the tubular epithelium via epithelial-mesenchyal transiti...
Journal ArticleDOI
Integrity of Cell-Cell Contacts Is a Critical Regulator of TGF-β1-Induced Epithelial-to-Myofibroblast Transition : Role for β-Catenin
András Masszi,Lingzhi Fan,László Rosivall,László Rosivall,Christopher A. McCulloch,Ori D. Rotstein,Istvan Mucsi,Andras Kapus +7 more
TL;DR: In this article, the authors investigated the role of β-catenin in TGF-β1-provoked epithelial-to-mesenchymal transition (EMT) in intact confluent monolayers.
Journal ArticleDOI
Complement activation by polyethoxylated pharmaceutical surfactants: Cremophor-EL, Tween-80 and Tween-20.
Zsoka Weiszhar,Judit Czúcz,Csaba Révész,László Rosivall,László Rosivall,Janos Szebeni,Zoltán Rozsnyay +6 more
TL;DR: Experimental evidences that polyethoxylated surfactants, Cremophor-EL and Tween-80, activate the complement system in vitro, in normal human serum and plasma are provided, consistent with the hypothesis that therapeutic side effects, such as acute hypersensitivity and systemic immunostimulation, can be attributed to complement activation-derived inflammatory mediators.
Journal ArticleDOI
Protective effects of mercaptoethylguanidine, a selective inhibitor of inducible nitric oxide synthase, in ligature-induced periodontitis in the rat
Zsolt Lohinai,Péter Benedek,Erzsébet Fehér,Adrienn Györfi,László Rosivall,Arpád Fazekas,Andrew L. Salzman,Csaba Szabó +7 more
TL;DR: It is proposed that enhanced formation of NO and peroxynitrite plays a significant role in the pathogenesis of periodontitis and that MEG treatment protects against the associated extravasation and bone destruction.