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Laura Fenster

Researcher at California Health and Human Services Agency

Publications -  75
Citations -  6752

Laura Fenster is an academic researcher from California Health and Human Services Agency. The author has contributed to research in topics: Pregnancy & Abortion. The author has an hindex of 45, co-authored 75 publications receiving 6428 citations. Previous affiliations of Laura Fenster include University of California, San Francisco & University of California, Berkeley.

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Exposure to Environmental and Mainstream Tobacco Smoke and Risk of Spontaneous Abortion

TL;DR: The authors examined the risk of spontaneous abortion from environmental tobacco smoke exposure in a prospective study of over 5,000 women conducted in California during 1990-1991 and found that the risks associated with ETS exposure were increased among nonsmokers who had moderate alcohol or heavy caffeine consumption.
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Residential exposure to traffic and spontaneous abortion.

TL;DR: In this article, the authors examined the effect of traffic exposure on adverse birth outcomes, such as low birth weight, and showed associations between air pollution or traffic exposure and adverse birth outcome.
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Caffeine Consumption and Menstrual Function

TL;DR: Caffeine intake was not strongly related to an increased risk for anovulation, short luteal phase, long follicular phase, short cycle length, long cycle, or measures of within-woman cycle variability.
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A study of the effect of perchloroethylene exposure on semen quality in dry cleaning workers.

TL;DR: The results suggest that occupational exposures to PCE can have subtle effects on sperm quality, and additional analyses are required to determine whether these effects are associated with changes in fertility.
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Assessment of reproductive disorders and birth defects in communities near hazardous chemical sites. III. Guidelines for field studies of male reproductive disorders.

TL;DR: The components of the ideal field study are summarized and research needs for improving field studies of male effects and for understanding the mechanisms of male reproductive toxicity are identified.