M
M. G. Nicholls
Researcher at University of Michigan
Publications - 9
Citations - 265
M. G. Nicholls is an academic researcher from University of Michigan. The author has contributed to research in topics: Angiotensin II & Epinephrine. The author has an hindex of 7, co-authored 9 publications receiving 263 citations.
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Journal ArticleDOI
Plasma norepinephrine variations with dietary sodium intake.
M. G. Nicholls,Wolfgang Kiowski,Wolfgang Kiowski,Andrew J. Zweifler,Stevo Julius,M. A. Schork,J Greenhouse +6 more
TL;DR: The findings show that sodium intake is an important consideration in the interpretation of plasma norepinephrine levels.
Journal ArticleDOI
Augmentation of the response to CRF in man: relative contributions of endogenous angiotensin and vasopressin.
Stella R. Milsom,J. V. Conaglen,Richard A. Donald,E. A. Espiner,M. G. Nicholls,John H. Livesey +5 more
TL;DR: AVP is likely to be of physiological importance in potentiating the ACTH response to CRF and that AVP and CRF together may provide a better test of pituitary ACTH secretion than either peptide alone.
Journal ArticleDOI
The effect of peripheral catecholamine concentrations on the pituitary-adrenal response to corticotrophin releasing factor in man.
TL;DR: Results show that modulation of peripheral plasma catecholamine levels within physiological limits does not affect CRF‐stimulated release of ACTH or the adrenal response in normal man.
Journal ArticleDOI
Evidence against an interaction of angiotensin ii with the sympathetic nervous system in man
TL;DR: Animal experiments indicate that angiotensin II can, under some circumstances stimulate the sympathetic nervous system at a number of different sites, and physiological levels of angiotENSin II are capable of interacting directly with the sympathetic system in man remains to be demonstrated.
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Effect of metoclopramide on aldosterone and regulatory factors in man
TL;DR: Results provide direct evidence that factors other than the plasma concentration of ACTH, All and K—or fluctuations in plasma catecholamines—are likely to be responsible for the acute increase in plasma aldosterone after metoclopramide injection in normal man.