M
M Oswald
Publications - 14
Citations - 1536
M Oswald is an academic researcher. The author has contributed to research in topics: COPD & Pulmonary hypertension. The author has an hindex of 11, co-authored 14 publications receiving 1473 citations.
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Journal ArticleDOI
Association of chronic obstructive pulmonary disease and sleep apnea syndrome
TL;DR: The association of chronic obstructive pulmonary disease (COPD) and sleep apnea syndrome (SAS) is likely to occur in a number of patients, and a large series of patients selected solely on the basis of a confirmed diagnosis of SAS are prospectively investigated.
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Pulmonary Hemodynamics in the Obstructive Sleep Apnea Syndrome: Results in 220 Consecutive Patients
TL;DR: In conclusion, PH is observed, in agreement with previous studies, in less than 20% of OSAS patients and is strongly linked to the presence of an obstructive (rather than restrictive) ventilatory pattern, hypoxemia, and hypercapnia.
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Daytime Pulmonary Hypertension in Patients with Obstructive Sleep Apnea Syndrome
Emmanuel Weitzenblum,Jean Krieger,Michel Apprill,Elisabeth Vallée,May Ehrhart,Julia Ratomaharo,M Oswald,Daniel Kurtz +7 more
TL;DR: Right heart catheterization was performed in a series of 46 consecutive patients in whom OSAS was firmly diagnosed by whole-night polysomnography, and there was no significant correlation between Ppa and the apnea index, the lowest sleep SaO2, or the time spent in apnea.
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Pulmonary hypertension in the obstructive sleep apnoea syndrome: prevalence, causes and therapeutic consequences.
TL;DR: Diurnal arterial blood gas disturbances and PH are most often explained by the presence of severe obesity (obesity-hypoventilation syndrome) and, principally, by association of OSA with chronic obstructive pulmonary disease (the so called "overlap syndrome").
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Pulmonary Hemodynamics in Patients With Chronic Obstructive Pulmonary Disease Before and During an Episode of Peripheral Edema
TL;DR: RVF is effectively present in at least some patients with COPD with peripheral edema and is associated with a significant increase of PAP from baseline, probably accounted for by hypoxic vasoconstriction, and pressure overload may contribute to the development of RVF.