M
Mali Mathru
Researcher at Loyola University Medical Center
Publications - 15
Citations - 549
Mali Mathru is an academic researcher from Loyola University Medical Center. The author has contributed to research in topics: Cardiac output & Pulmonary wedge pressure. The author has an hindex of 10, co-authored 15 publications receiving 538 citations.
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Journal ArticleDOI
Norepinephrine and Phenylephrine Effects on Right Ventricular Function in Experimental Canine Pulmonary Embolism
TL;DR: It is suggested that in the clinical setting of acute PE, the judicious use of NE, rather than PHEN, may be more beneficial in restoring RV function and systemic hemodynamics.
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Permeability pulmonary edema following lung resection.
TL;DR: The etiology of edema associated with pulmonary resection was investigated in five patients during the immediate postoperative period, finding an increase in pulmonary capillary pressure associated with passage of a normal to high cardiac output in a reduced volume pulmonary vascular bed to be significant.
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Urine Hydrogen Peroxide During Adult Respiratory Distress Syndrome in Patients With and Without Sepsis
Mali Mathru,Michael W. Rooney,David J. Dries,Leroy J. Hirsch,Lionel Barnes,Martin J. Tobin,Martin J. Tobin +6 more
TL;DR: These studies demonstrate that urine H2O2 may be a useful analyte to differentiate the severity of illness in patients with or nonsurvivor ARDS and with sepsis, and that ARDS alone is the major contributor to the H 2O2 oxidant processes.
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Pressure support. Changes in ventilatory pattern and components of the work of breathing.
W. B. Van De Graaff,K. Gordey,S. E. Dornseif,David J. Dries,Bruce Kleinman,Pankaj Kumar,Mali Mathru +6 more
TL;DR: It is found that increasing the pressure support level (PSL) did not change minute ventilation, PCO2, or pH despite large changes in both rate and depth of breathing, and total inspiratory WOB per minute increased by 186 +/- 29 percent, demonstrating that PS results in a respiratory pattern requiring substantially greater total mechanical work.
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Effect of Fast vs Slow Intralipid Infusion on Gas Exchange, Pulmonary Hemodynamics, and Prostaglandin Metabolism
TL;DR: It was unable to demonstrate a cause and effect relationship between plasma levels of 6-keto-PGF1 alpha and thromboxane B2 and the observed pulmonary hemodynamic response to slow or fast Intralipid infusion.