M
Mami Yamada
Researcher at Nagoya City University
Publications - 4
Citations - 56
Mami Yamada is an academic researcher from Nagoya City University. The author has contributed to research in topics: Endothelial stem cell & Skeletal muscle. The author has an hindex of 3, co-authored 4 publications receiving 29 citations.
Papers
More filters
Journal ArticleDOI
p62/SQSTM1 and Nrf2 are essential for exercise-mediated enhancement of antioxidant protein expression in oxidative muscle.
TL;DR: It is demonstrated that muscle contractile activity increases antioxidants, Nrf2 translocation into nuclei, and NRF2 DNA‐binding activity in association with increased p62 phosphorylation (Ser351) in mouse oxidative skeletal muscle, indicating that p62 and N RF2 cooperatively regulate the exercise‐mediated increase of antioxidants in oxidative muscle.
Journal ArticleDOI
Muscle-derived SDF-1α/CXCL12 modulates endothelial cell proliferation but not exercise training-induced angiogenesis
Mami Yamada,Chihiro Hokazono,Ken Tokizawa,Shuri Marui,Masahiro Iwata,Vitor A. Lira,Katsuhiko Suzuki,Shinji Miura,Kei Nagashima,Mitsuharu Okutsu +9 more
TL;DR: It is postulated that the chemokine stromal cell-derived factor-1 (SDF-1α/CXCL12), shown to promote neovascularization in several organs, contributes to angiogenesis in skeletal muscle and is abundantly expressed in capillary-rich oxidative soleus and exercise-trained plantaris muscles.
Journal ArticleDOI
Maternal exercise training attenuates endotoxin-induced sepsis in mice offspring.
TL;DR: Maternal exercise inhibited the LPS-induced inflammatory response in female offspring, suggesting that regular exercise during pregnancy could be a potential candidate of the onset of sepsis and MODS in offspring.
Journal ArticleDOI
Regular exercise stimulates endothelium autophagy via IL-1 signaling in ApoE deficient mice
Mitsuharu Okutsu,Mami Yamada,Ken Tokizawa,Shuri Marui,Katsuhiko Suzuki,Vitor A. Lira,Kei Nagashima +6 more
TL;DR: This article showed that 16 weeks of voluntary exercise reduced high-fat diet-induced atherosclerotic plaque formation in the aortic root of ApoE deficient mice, and that this protection occurred without changes in circulating triglycerides, total cholesterol, and lipoproteins.