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Maria Paola Bertuccio

Researcher at University of Messina

Publications -  24
Citations -  366

Maria Paola Bertuccio is an academic researcher from University of Messina. The author has contributed to research in topics: Medicine & Apoptosis. The author has an hindex of 10, co-authored 18 publications receiving 244 citations.

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Vaccine hesitancy: An overview on parents' opinions about vaccination and possible reasons of vaccine refusal

TL;DR: Data analysis shows that parents are, theoretically, favourable towards vaccinations but have little knowledge of such practices, sometimes not being unaware of the types of vaccines administrated to their children, and that health education of parents is the cornerstone on which the public health should build the fight to the vaccine hesitancy and improve the vaccination coverages.
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Toxicological assessment of multi-walled carbon nanotubes on A549 human lung epithelial cells.

TL;DR: Based on the physical and chemical features of MWCNTs, several toxicological pathways could be activated in respiratory epithelium upon their inhalation and the biological impacts of nano-needles were imputable to their efficient and very fast uptake and to the resulting mechanical damages in cell compartments.
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In vitro assessment of neurotoxicity and neuroinflammation of homemade MWCNTs.

TL;DR: The results underlined the potential onset of neurodegenerative diseases, due to unintentional MWCNT exposure, and the almost superimposable pro-oxidant activity of both CNTs could be imputable to excessive lengths with regard to the pristine M WCNTs and to the eroded surface, causing increased reactivity.
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Higher levels of oxidative DNA damage in cervical cells are correlated with the grade of dysplasia and HPV infection.

TL;DR: The authors' results showed greater oxidative damage in HPV‐related dysplastic cervical lesions compared to samples with normal cytology, especially in women with high‐grade squamous intraepithelial lesions, which could facilitate the integration of the virus, promoting HPV carcinogenesis.
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Mitochondrial-mediated apoptosis pathway in alveolar epithelial cells exposed to the metals in combustion-generated particulate matter.

TL;DR: Results improve the knowledge of the pathogenic role that airborne metals and in particular V exerted in respiratory epithelium and specifically p53 involvement and metabolic impairment through changes in HIF-1α and Glut-1 expression were determined.