M
Maria Shabbir
Researcher at Quaid-i-Azam University
Publications - 43
Citations - 1255
Maria Shabbir is an academic researcher from Quaid-i-Azam University. The author has contributed to research in topics: Biology & Medicine. The author has an hindex of 11, co-authored 21 publications receiving 934 citations. Previous affiliations of Maria Shabbir include National University of Sciences and Technology & University of Wisconsin-Madison.
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Antioxidant activity, total phenolic and total flavonoid contents of whole plant extracts Torilis leptophylla L
TL;DR: Data from present results revealed that Torilis leptophylla act as an antioxidant agent due to its free radical scavenging and cytoprotective activity.
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Assessment of phytochemicals, antioxidant, anti-lipid peroxidation and anti-hemolytic activity of extract and various fractions of Maytenus royleanus leaves
TL;DR: The therapeutic potential of Maytenus royleanus leaves, in particular, methanol extract, ethyl acetate and n-butanol fraction as therapeutic agent against free-radical associated damages is suggested.
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MicroRNAs in skin response to UV radiation.
TL;DR: This review summarizes the current knowledge of the role of miRNAs in the regulation of the human skin response upon exposure to UV radiation.
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Antioxidant activity of polyphenolic compounds isolated from ethyl-acetate fraction of Acacia hydaspica R. Parker
TL;DR: The antioxidant activity of polyphenolic compounds of A. hydaspica merit further investigations for consumption of this plant in oxidative stress related disorders and the isolation of catechin from this new species could provide a varied opportunity to obtain large quantities of cateschine and cATEchin isomers beside from green tea.
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Centriole Overduplication is the Predominant Mechanism Leading to Centrosome Amplification in Melanoma
Ryan A. Denu,Maria Shabbir,Minakshi Nihal,Chandra K. Singh,B. Jack Longley,B. Jack Longley,Mark E. Burkard,Nihal Ahmad,Nihal Ahmad +8 more
TL;DR: It was demonstrated that centriole overduplication is the predominant mechanism leading to centrosome amplification in melanoma and that PLK4 should be further evaluated as a potential therapeutic target for melanoma treatment.