scispace - formally typeset
M

Mark W. Cowan

Researcher at Azul Systems

Publications -  29
Citations -  2771

Mark W. Cowan is an academic researcher from Azul Systems. The author has contributed to research in topics: Transducer & Ultrasound energy. The author has an hindex of 20, co-authored 28 publications receiving 2768 citations.

Papers
More filters
Patent

Leadless tissue stimulation systems and methods

TL;DR: In this paper, a controller-transmitter is activated at a remote tissue location to transmit/deliver acoustic energy through the body to a receiver-stimulator at a target tissue location.
Patent

Therapeutic ultrasonic catheter for delivering a uniform energy dose

TL;DR: A catheter (30) is a method for treating a target region in a body lumen comprising directing a uniform dose of ultrasonic energy from an interior of the lumen, wherein the dosage of ultrasound energy received at any one point along the length varies by no more than plus or minus 6 decibels as discussed by the authors.
Patent

Optimizing energy transmission in a leadless tissue stimulation system

TL;DR: In this paper, a method and systems for optimizing acoustic energy transmission in implantable devices are disclosed. But the authors focus on the location of the receiver and the efficiency of the transmitted acoustic beam received by the receiver.
Patent

Systems and methods for implantable leadless tissue stimulation

TL;DR: In this paper, the authors used electrical stimulation of the spine, where vibrational energy from a source is received by an implanted device and converted to electrical energy and the converted electrical energy is used by implanted electrodes to stimulate the pre-determined brain site.
PatentDOI

Methods and apparatus for uniform transcutaneous therapeutic ultrasound

TL;DR: In this paper, a wide beam ultrasound delivery system providing a uniform exposure field is used to enhance the uptake of injected substances and/or enhance the transfection of DNA in the tissues of human subject, or reduce the amount of vascular intimal hyperplasia in human subjects following vascular injury.