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Masood Akhtar

Researcher at University of Wisconsin-Madison

Publications -  233
Citations -  8626

Masood Akhtar is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Tachycardia & Ventricular tachycardia. The author has an hindex of 47, co-authored 232 publications receiving 8491 citations. Previous affiliations of Masood Akhtar include Wisconsin Alumni Research Foundation & Medtronic plc.

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Automatic Implantable Cardioverter Defibrillators and Survival of Patients with Left Ventricular Dysfunction and Malignant Ventricular Arrhythmias

TL;DR: The automatic implantable cardioverter defibrillator is probably highly effective in preventing arrhythmic mortality even in high-risk patients and such treatment does not appear to significantly impair a patient's functional status.
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Sustained bundle branch reentry as a mechanism of clinical tachycardia.

TL;DR: The data suggest that sustained BBR is not an uncommon mechanism of tachycardia; it can be induced readily in the laboratory and is amendable to catheter ablation by the very nature of its circuit.
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Atrioverter: An Implantable Device for the Treatment of Atrial Fibrillation

TL;DR: With the Atrioverter, prompt and safe restoration of sinus rhythm is possible in patients with recurrent atrial fibrillation, and this system was evaluated in a prospective, multicenter study.
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Adenosine for paroxysmal supraventricular tachycardia: dose ranging and comparison with verapamil. Assessment in placebo-controlled, multicenter trials. The Adenosine for PSVT Study Group.

TL;DR: Adenosine in graded doses up to 12 mg rapidly and effectively terminates acute episodes of paroxysmal supraventricular tachycardia in which the atrioventricular node is an integral part of the re-entrant circuit.
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Comparison of Cardiac Pacing with Drug Therapy in the Treatment of Neurocardiogenic (Vasovagal) Syncope with Bradycardia or Asystole

TL;DR: In patients with neurocardiogenic syncope associated with bradycardia or asystole, drug therapy is often effective in preventing syncope, whereas artificial pacing is not.