M
Matthias Rohleder
Researcher at Ludwig Maximilian University of Munich
Publications - 13
Citations - 692
Matthias Rohleder is an academic researcher from Ludwig Maximilian University of Munich. The author has contributed to research in topics: Vitrectomy & Macular hole. The author has an hindex of 10, co-authored 13 publications receiving 642 citations. Previous affiliations of Matthias Rohleder include Moorfields Eye Hospital.
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Journal ArticleDOI
Posterior vitreous detachment induced by microplasmin.
Arnd Gandorfer,Matthias Rohleder,Matthias Rohleder,C S Sethi,Dominik Eckle,Ulrich Welge-Lussen,Anselm Kampik,Philip J. Luthert,David G. Charteris +8 more
TL;DR: Both after death and in vivo, microplasmin induces a dose-dependent cleavage between the vitreous cortex and the ILM without morphologic alterations of the retina in the feline eye.
Journal ArticleDOI
Epiretinal pathology of vitreomacular traction syndrome.
TL;DR: There are two distinct clinicopathological features of vitreomacular traction syndrome which suggest different forms of epiretinal fibrocellular proliferation, which may help to explain the high prevalence of cystoid macular oedema and progressive vitreOMacular traction characteristic for this disorder.
Journal ArticleDOI
Epiretinal pathology of diffuse diabetic macular edema associated with vitreomacular traction.
Arnd Gandorfer,Matthias Rohleder,Sabine Grosselfinger,Christos Haritoglou,Michael W. Ulbig,Anselm Kampik +5 more
TL;DR: The vitreomacular interface in eyes with DDME is characterized by a layer of native vitreous collagen and a varying cellular component and Resolution of macular edema does not depend on the presence and removal of contractile membranes.
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Ultrastructure of the vitreomacular interface in full-thickness idiopathic macular holes: a consecutive analysis of 100 cases.
Ricarda G. Schumann,Markus Schaumberger,Matthias Rohleder,Christos Haritoglou,Anselm Kampik,Arnd Gandorfer +5 more
TL;DR: Incomplete vitreoretinal separation may contribute to the development of epimacular membranes in eyes with idiopathic macular holes and fibrocellular proliferation appears to be a secondary event instead of a primary feature in macular hole development.
Journal ArticleDOI
Idiopathic macular holes: ultrastructural aspects of surgical failure.
Ricarda G. Schumann,Matthias Rohleder,Markus Schaumberger,Christos Haritoglou,Anselm Kampik,Arnd Gandorfer +5 more
TL;DR: Eyes with idiopathic macular holes that were found not to be closed early after the first vitrectomy show massive proliferation of cells and newly formed collagen irregularly distributed at the remaining ILM after surgical intervention.