M
Matthias Stöck
Researcher at Leibniz Association
Publications - 111
Citations - 3325
Matthias Stöck is an academic researcher from Leibniz Association. The author has contributed to research in topics: Brown adipose tissue & Thermogenesis. The author has an hindex of 31, co-authored 105 publications receiving 2809 citations. Previous affiliations of Matthias Stöck include Martin Luther University of Halle-Wittenberg & University of Lausanne.
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Journal ArticleDOI
Sex Chromosome Conservation, DMRT1 Phylogeny and Gonad Morphology in Diploid Palearctic Green Toads (Bufo viridis Subgroup)
Stephanie Tamschick,Beata Rozenblut-Kościsty,Lucio Bonato,Christophe Dufresnes,Petros Lymberakis,Werner Kloas,Maria Ogielska,Matthias Stöck +7 more
TL;DR: 3 cross-amplifying sex-linked microsatellite markers are used to uncover sex determination systems and sex chromosomes in purebred, diploid Palearctic green toads (Bufo viridis subgroup), which had so far only been characterized in laboratory-bred hybrids, and support an XY system in B. viridis and B. variabilis.
Journal ArticleDOI
Influence of subdiaphragmatic vagotomy and brown fat sympathectomy on thermogenesis in rats
TL;DR: The data demonstrate that the changes in brown fat activity induced by exogenous insulin are mediated by the sympathetic nervous system and that the depressed thermogenesis and brownfat activity associated with vagotomy appear to be due to a relative insulin deficiency and can be reversed by treatment with the hormone.
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Thirteen polymorphic microsatellite markers for the European green toad Bufo viridis viridis, a declining amphibian species
TL;DR: 13 new polymorphic microsatellite markers for the European green toad Bufo viridis viridis (B. viridis subgroup), a declining amphibian from Central, Southeastern and Eastern Europe, are reported.
Journal Article
Thermogenic properties of ciclazindol and mazindol in rodents.
TL;DR: Some of the physiological and behavioural effects of ciclazindol and mazINDol analogues in rats are examined, including an increase in RMR that cannot be dissociated from central stimulant actions.