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Matthias Sturzenegger

Researcher at University of Bern

Publications -  128
Citations -  7811

Matthias Sturzenegger is an academic researcher from University of Bern. The author has contributed to research in topics: Whiplash & Stroke. The author has an hindex of 50, co-authored 127 publications receiving 7405 citations. Previous affiliations of Matthias Sturzenegger include University of Washington & University Hospital of Bern.

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Long-term outcome after whiplash injury. A 2-year follow-up considering features of injury mechanism and somatic, radiologic, and psychosocial findings.

TL;DR: According to a strict definition of whiplash injury, a consecutive nonselected sample of 117 patients with recent injury who had similar sociocultural and educational backgrounds were assessed, finding symptomatic patients scored higher with regard to impaired well-being and performed worse on tasks of attentional functioning.
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Vertebral Artery Dissection Presenting Findings and Predictors of Outcome

TL;DR: Favorable outcome is observed in most ischemic strokes and independently predicted by low National Institutes of Health Stroke Scale score and younger age.
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Ischaemic stroke in young adults: predictors of outcome and recurrence

TL;DR: Severe neurological deficits at presentation, total anterior circulation stroke, and diabetes mellitus predict unfavourable outcome, and previous TIA are associated with increased risk of recurrence.
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Encephalopathy associated with Hashimoto thyroiditis: Diagnosis and treatment

TL;DR: Six patients with Hashimoto thyroiditis and associated encephalopathy are described and compared and it is shown that two types of initial clinical presentation can be differentiated: a vasculitic type with stroke-like episodes and mild cognitive impairment in nine patients, and a diffuse progressive type with dementia, seizures, psychotic episodes or altered consciousness in 11 patients.
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Carotid dissection with and without ischemic events Local symptoms and cerebral artery findings

TL;DR: Data suggest that ICAD causing high-grade stenosis and occlusion are more likely to lead to intracranial obstructions and cerebral or retinal ischemic events, and Conversely, ICAD without luminal narrowing cause more local signs and symptoms.