M
Max Nedelmann
Researcher at University of Hamburg
Publications - 4
Citations - 581
Max Nedelmann is an academic researcher from University of Hamburg. The author has contributed to research in topics: Staphylococcus epidermidis & Mutant. The author has an hindex of 4, co-authored 4 publications receiving 563 citations.
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Characterization of transposon mutants of biofilm-producing Staphylococcus epidermidis impaired in the accumulative phase of biofilm production: genetic identification of a hexosamine-containing polysaccharide intercellular adhesin.
TL;DR: The results demonstrate that the mutants were impaired in the accumulative phase of biofilm production by S. epidermidis by mediating intercellular adhesion.
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Identification of Three Essential Regulatory Gene Loci Governing Expression of Staphylococcus epidermidis Polysaccharide Intercellular Adhesin and Biofilm Formation
Dietrich Mack,Holger Rohde,Sabine Dobinsky,Joachim Riedewald,Max Nedelmann,Johannes K.-M. Knobloch,Holger-A. Elsner,Hubert Feucht +7 more
TL;DR: The formation of adherent multilayered biofilms embedded into a glycocalyx represents an essential factor in the pathogenesis of Staphylococcus epidermidis biomaterial-related infections.
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Establishment of an arbitrary PCR for rapid identification of Tn917 insertion sites in Staphylococcus epidermidis: characterization of biofilm-negative and nonmucoid mutants.
Johannes K.-M. Knobloch,Max Nedelmann,Kathrin Kiel,Katrin Bartscht,Matthias A. Horstkotte,Sabine Dobinsky,Holger Rohde,Dietrich Mack +7 more
TL;DR: An arbitrary PCR was established for the rapid and easy identification of Tn917 insertion sites in Staphylococcus epidermidis with six independent, well-characterized biofilm-negative Tn 917 transposon mutants, which were clustered in the icaADBC gene locus or harbor Tn717 in the regulatory gene rsbU.
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Generalized transduction for genetic linkage analysis and transfer of transposon insertions in different Staphylococcus epidermidis strains.
TL;DR: S. epidermidis phage 71 was used to prove genetic linkage between transposon insertion and altered phenotype by generalized transduction, and will be a useful tool facilitating the study of bacterial determinants of the pathogenicity of S. epidersmidis.