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Michael D. Martin

Researcher at University of Oklahoma Health Sciences Center

Publications -  19
Citations -  289

Michael D. Martin is an academic researcher from University of Oklahoma Health Sciences Center. The author has contributed to research in topics: Myelopathy & Medicine. The author has an hindex of 5, co-authored 19 publications receiving 201 citations. Previous affiliations of Michael D. Martin include University of Oklahoma.

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Pathophysiology of lumbar disc degeneration: a review of the literature

TL;DR: Lumbar disc degeneration occurs because of a variety of factors and results in a multitude of conditions, including herniation with radiculopathy and chronic discogenic pain.
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Regenerative rehabilitation with conductive biomaterials for spinal cord injury

TL;DR: Current regenerative medicine approaches in clinical trials and the rehabilitation, or neuromodulation, approaches for SCI, are discussed, along with their respective translational limitations, and the translational potential, in a surgical context, of conductive biomaterials as they pertain to SCI.
Journal Article

Differential expression of type 2 3α/type 5 17β-hydroxysteroid dehydrogenase (AKR1C3) in tumors of the central nervous system.

TL;DR: A widespread expression of AKR1C3 is described in glial neoplasms and meningiomas, with limited expression in medulloblastoma and no expression in Schwannoma, corroborate earlier observations that AKR 1C3 was expressed in both sex hormone-dependent and hormone-independent malignancies.
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Surgical Treatment of Tethered Cord Syndrome in Adults: A Systematic Review and Meta-Analysis.

TL;DR: Tethered cord syndrome should be included in the differential diagnosis in patients presenting with back or leg pain, somatosensory symptoms of the lower extremities, muscular weakness, urodynamic dysfunction, or bowel dysfunction, and patients should be counseled about surgical de-tethering as an option.
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Spinal cord detethering in children with tethered cord syndrome and Chiari type 1 malformations.

TL;DR: Pediatric patients who have both CM1 and TCS, but do not demonstrate classic Chiari-related symptoms, may experience symptomatic improvement after filum terminale sectioning.