M
Millet Vg
Publications - 8
Citations - 79
Millet Vg is an academic researcher. The author has contributed to research in topics: Essential hypertension & Nephrotic syndrome. The author has an hindex of 6, co-authored 8 publications receiving 77 citations.
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Journal ArticleDOI
Control of hypertension with the angiotensin converting enzyme inhibitor captopril reduces glomerular proteinuria.
TL;DR: Captopril supports the concept that captopril may have salutary effects on the glomerulus, independently of its effect on systemic blood pressure.
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Partial deficit of hypoxanthine guanine phosphoribosyl transferase presenting as acute renal failure.
TL;DR: An enzymatic study showed the existence of a partial hypoxanthine-guanine phosphoribosyl transferase (HPRT) deficit (less than 1% of HPRT normal activity); this possibility should be considered whenever an ARF is accompanied by disproportionate high levels of serum uric acid.
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The role of urinary PGE2 and renin-angiotensin-aldosterone system in the pathogenesis of essential hypertension.
Luis M. Ruilope,R. Garcia Robles,Barrientos A,C. Bernis,Jose M. Alcazar,J. A.F. Tresguerres,Mancheño E,Millet Vg,Jose M. Sancho,Jose L. Rodicio +9 more
TL;DR: LREH and NREH may be regarded as two different populations distinguishable not only by different secretion of PRA but also by different excretion of PGE2 in urine and by their characteristic response to indomethacin.
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Improvement of Anaemia With Desferrioxamine in Haemodialysis Patients
Manuel Praga,Amado Andrés,J de la Serna,Luis M. Ruilope,J Nieto,J Estenoz,Millet Vg,F Arnaiz,Jose L. Rodicio +8 more
TL;DR: DFO therapy should be considered in haemodialysis patients with severe anaemia and increased blood transfusion requirements, as the tolerance to DFO was excellent.
Journal ArticleDOI
Acute Renal Failure and Tubular Dysfunction Associated with Minimal Change Nephrotic Syndrome
Manuel Praga,Magaly Martinez,Amado Andrés,M. Alvarez de Buergo,Bello I,Millet Vg,J. Ma. Morales +6 more
TL;DR: A case of minimal change nephrotic syndrome in which acute renal function derangement developed in the absence of any precipitating factor is reported.