N
Nadima S. Shegem
Researcher at University of Jordan
Publications - 12
Citations - 301
Nadima S. Shegem is an academic researcher from University of Jordan. The author has contributed to research in topics: Diabetes mellitus & Medicine. The author has an hindex of 7, co-authored 9 publications receiving 280 citations.
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Journal ArticleDOI
Sexual Dysfunction in Jordanian Diabetic Women
TL;DR: Diabetic women had more dysfunction of desire, arousal, lubrication, and orgasm than nondiabetic women, and age, BMI, duration of diabetes, and the presence of coronary artery disease, nephropathy, and retinopathy had negative effects on FSD.
Journal Article
Prevalence and predictors of diabetic foot syndrome in type 2 diabetes mellitus in Jordan
AbdulKareem S. Jbour,Nadim Jarrah,Abdelrahman M Radaideh,Nadima S. Shegem,Ismail M. Bader,Anwar Batieha,Kamel Ajlouni +6 more
TL;DR: Prevalence of amputation correlates with duration of diabetes, poor glycemic control, smoking, neurological impairment, peripheral vascular disease and microalbuminuria.
Journal Article
Sexual dysfunction among Jordanian men with diabetes.
Faisal A. Khatib,Nadim S. Jarrah,Nadima S. Shegem,Anwar M. Bateiha,Ruba M. Abu-Ali,Kamel Ajlouni +5 more
TL;DR: Prevalence of erectile dysfunction among Jordanian diabetic patients is high and increases with age and poor glycemic control, and other independent risk factors include: hypertension, coronary artery disease, retinopathy and neuropathy.
Journal ArticleDOI
Clinical and inheritance profiles of Kallmann syndrome in Jordan.
TL;DR: The majority of cases represented the X-linked form of KS, which might point to a high prevalence of Kal 1 gene in the population, and genetic counselling helps families to reach a diagnosis at an early age and to decide about their reproductive options.
Journal Article
Effects of short term metformin administration on androgens in normal men.
Nadima S. Shegem,Abeer M. Alsheek Nasir,Abdel-Kareem Jbour,Anwar Batieha,M El-Khateeb,Kamel Ajlouni +5 more
TL;DR: Metformin administration was associated with a reduction in total testosterone, free testosterone, and 17-hydroxyprogesterone and an increase in sex hormone binding globulin and dehydroepiandrosterone sulphate in normal males.