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Oana M. Dumitrascu

Researcher at Cedars-Sinai Medical Center

Publications -  59
Citations -  612

Oana M. Dumitrascu is an academic researcher from Cedars-Sinai Medical Center. The author has contributed to research in topics: Medicine & Engineering. The author has an hindex of 10, co-authored 39 publications receiving 316 citations. Previous affiliations of Oana M. Dumitrascu include Johns Hopkins University School of Medicine & Mayo Clinic.

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Identification of early pericyte loss and vascular amyloidosis in Alzheimer’s disease retina

TL;DR: The identification of pericyte and PDGFRβ loss accompanying increased vascular amyloidosis in Alzheimer’s retina implies compromised blood–retinal barrier integrity and provides new targets for AD diagnosis and therapy.
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Still cooling after all these years: Meta-analysis of pre-clinical trials of therapeutic hypothermia for acute ischemic stroke.

TL;DR: Using several outcome measures in a variety of models and species, therapeutic hypothermia was protective compared with normothermia, with powerful and statistically significant normalized treatment effect sizes, in 60 papers comprising 216 comparisons.
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Acute ophthalmic artery occlusion in a COVID-19 patient on apixaban.

TL;DR: A case of ophthalmic artery occlusion (OAO) in a young patient with Covid-19 infection that was on therapeutic anticoagulation with apixaban for deep venous thrombosis (DVT), which prompted further investigations and upper and lower extremities DVTs were confirmed and managed with enoxaparin.
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Thrombolysis for Central Retinal Artery Occlusion in 2020: Time Is Vision!

TL;DR: Future research should focus on developing feasible retinal penumbra imaging studies that, similar to cerebral tissue viability or perfusion imaging, can be incorporated into the thrombolysis decision-making algorithm.
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Retinal Microvascular Abnormalities as Surrogate Markers of Cerebrovascular Ischemic Disease: A Meta-Analysis.

TL;DR: Focal arteriolar narrowing and retinopathy predicted CVD subtypes after risk-factor adjustment, suggesting that features different than traditional vascular risk factors, are involved in CVD pathophysiology.