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Osama Hamdy

Researcher at Harvard University

Publications -  128
Citations -  6391

Osama Hamdy is an academic researcher from Harvard University. The author has contributed to research in topics: Diabetes mellitus & Type 2 diabetes. The author has an hindex of 29, co-authored 108 publications receiving 5371 citations. Previous affiliations of Osama Hamdy include Beth Israel Deaconess Medical Center & Mansoura University.

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Prevention and management of type 2 diabetes: dietary components and nutritional strategies

TL;DR: With an emphasis on overall diet quality, several dietary patterns such as Mediterranean, low glycaemic index, moderately low carbohydrate, and vegetarian diets can be tailored to personal and cultural food preferences and appropriate calorie needs for weight control and diabetes prevention and management.
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Testosterone, sex hormone-binding globulin, and the development of type 2 diabetes in middle-aged men: prospective results from the Massachusetts male aging study.

TL;DR: The prospective findings are consistent with previous, mainly cross-sectional reports, suggesting that low levels of testosterone and SHBG play some role in the development of insulin resistance and subsequent type 2 diabetes.
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Metabolic obesity: the paradox between visceral and subcutaneous fat.

TL;DR: The term "Metabolic Obesity", in reference to visceral fat accumulation in either lean or obese individuals may identify those at risk for cardiovascular disease better than the currently used definitions of obesity.
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Effect of lifestyle modification on adipokine levels in obese subjects with insulin resistance.

TL;DR: Weight reduction in obese individuals with insulin resistance was associated with a significant decrease in leptin and IL-6 and a tendency toward a decrease in circulating TNF-alpha, whereas adiponectin was increased only in diabetic subjects.
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Lifestyle Modification Improves Endothelial Function in Obese Subjects With the Insulin Resistance Syndrome

TL;DR: It is concluded that 6 months of weight reduction and exercise improve macrovascular endothelial function and reduces selective markers of endothelial activation and coagulation in obese subjects with IRS regardless of the degree of glucose tolerance.