P. Tammaro

TL;DR: Anorexia nervosa caused demonstrable abnormalities of mitral valve motion and reduced left ventricular mass and filling associated with systolic dysfunction.

TL;DR: Results clearly indicate that early abnormalities of cardiac structure and function are observed not only in patients with NIDDM, but also in those with impaired glucose tolerance, independent of the confounding role of myocardial ischemia, body weight, and blood pressure.

TL;DR: The superior LVM regression with tel Misartan versus carvedilol suggests telmisartan has a mechanism that may be beyond that of lowering BP in hypertensive patients.

TL;DR: Early cardiac alterations and a tendency for effort hypertension in children of hypertensives are confirmed and could be explained by effort systolic overload or by a common response to an increased adrenergic stimulus.

TL;DR: Adult patients with mild-to-moderate hypertension and an optimal acoustic window by two-dimensional echocardiography were randomised at baseline to 12 months' double-blind, once-daily treatment with telmisartan, an angiotensin II receptor blocker, and tolerability and effects on left ventricular mass index were compared with HCTZ.