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Paul E. Milbury

Researcher at United States Department of Agriculture

Publications -  19
Citations -  2347

Paul E. Milbury is an academic researcher from United States Department of Agriculture. The author has contributed to research in topics: Antioxidant & Metabolome. The author has an hindex of 15, co-authored 19 publications receiving 2221 citations. Previous affiliations of Paul E. Milbury include University of California, San Francisco & University of Texas at Austin.

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Flavonoid-rich dark chocolate improves endothelial function and increases plasma epicatechin concentrations in healthy adults.

TL;DR: Flavonoid-rich dark chocolate improves endothelium-dependent flow-mediated dilation of the brachial artery and is associated with an increase in plasma epicatechin concentrations in healthy adults.
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Kynurenine pathway abnormalities in Parkinson's disease

TL;DR: 3-O-methyldopa (3OMD) concentration appears to be a reliable marker of L-dopa therapy, and may be a further indication of a defect in NADH:ubiquinone oxidoreductase (complex I) in mitochondria in Parkinson's disease (PD).
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Flavonoids from Almond Skins Are Bioavailable and Act Synergistically with Vitamins C and E to Enhance Hamster and Human LDL Resistance to Oxidation

TL;DR: Almond skin flavonoids possess antioxidant capacity in vitro; they are bioavailable and act in synergy with vitamins C and E to protect LDL against oxidation in hamsters.
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Determination of flavonoids and phenolics and their distribution in almonds.

TL;DR: Total phenols, flavonoids, and phenolic acids in California almond skins and kernels among the principal almond varieties are determined with high-performance liquid chromatography/tandem mass spectrometry under identical HPLC conditions to lead to a better understanding of the mechanisms of action underlying the relationship between almond consumption and health-related outcomes.
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Kynurenic acid concentrations are reduced in Huntington's disease cerebral cortex.

TL;DR: Both uric acid and glutathionine were significantly reduced in several regions of HD cerebral cortex, which could signify abnormal energy metabolism in HD, and kynurenic acid is an antagonist of excitatory amino acid receptors, a deficiency could contribute to the pathogenesis of neuronal degeneration in HD.