Osteoblast adhesion on biomaterials.

http://www.endoexperience.com/documents/Cellularmolecularandtissuelevelreactionstoorthoforce_2006.pdf

Cellular, molecular, and tissue-level reactions to orthodontic force

The pathogenesis of periodontitis involves a complex immune/inflammatory cascade that is initiated by the bacteria of the oral biofilm that forms naturally on the teeth. The susceptibility to periodontitis appears to be determined by the host response; specifically, the magnitude of the inflammatory response and the differential activation of immune pathways. The purpose of this review was to delineate our current knowledge of the host response in periodontitis. The role of innate immunity, the failure of acute inflammation to resolve (thus becoming chronic), the cytokine pathways that regulate the activation of acquired immunity and the cells and products of the immune system are considered. New information relating to regulation of both inflammation and the immune response will be reviewed in the context of susceptibility to, and perhaps control of, periodontitis.

/pdf/inflammatory-and-immune-pathways-in-the-pathogenesis-of-4igej65lbb.pdf

Inflammatory and immune pathways in the pathogenesis of periodontal disease.

Techniques for mechanical stimulation of cells in vitro: a review

The optic nerve head in glaucoma: role of astrocytes in tissue remodeling

Perception of discomfort by patients undergoing orthodontic treatment

Interleukin 1 beta and prostaglandin E are involved in the response of periodontal cells to mechanical stress in vivo and in vitro

Diagnosis and treatment of open bite malocclusion challenges pediatric dentists who attempt to intercept this malocclusion at an early age. This article updates clinicians on the causes and cures of anterior open bite based on clinical data. Patients with open bite malocclusion can be diagnosed clinically and cephalometrically, however, diagnosis should be viewed in the context of the skeletal and dental structure. Accurate classification of this malocclusion requires experience and training. Simple open bite during the exchange of primary to permanent dentition usually resolves without treatment. Complex open bites that extend farther into the premolar and molar regions, and those that do not resolve by the end of the mixed dentition years may require orthodontic and/or surgical intervention. Vertical malocclusion develops as a result of the interaction of many different etiologic factors including thumb and finger sucking, lip and tongue habits, airway obstruction, and true skeletal growth abnormalities. Treatment for open bite ranges from observation or simple habit control to complex surgical procedures. Successful identification of the etiology improves the chances of treatment success. Vertical growth is the last dimension to be completed, therefore treatment may appear to be successful at one point and fail later. Some treatment may be prolonged, if begun early. Long-term clinical outcomes are needed to determine treatment effectiveness and clinicians should consider the cost-effectiveness of these early initiated and protracted plans.

Open bite: a review of etiology and management.

https://dentistry.hsc.wvu.edu/media/1182/treatment-response-and-long-term-dentofacial-adaptations-to-maxillary-expansion-and-protraction.pdf

Peter Ngan

Papers

Perception of discomfort by patients undergoing orthodontic treatment

Interleukin 1 beta and prostaglandin E are involved in the response of periodontal cells to mechanical stress in vivo and in vitro

Open bite: a review of etiology and management.

Treatment response and long-term dentofacial adaptations to maxillary expansion and protraction.

Soft tissue and dentoskeletal profile changes associated with maxillary expansion and protraction headgear treatment