P
Preeti Srivastava
Researcher at National Institutes of Health
Publications - 6
Citations - 264
Preeti Srivastava is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Gene & Origin recognition complex. The author has an hindex of 6, co-authored 6 publications receiving 250 citations.
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Journal ArticleDOI
Multipartite Regulation of rctB, the Replication Initiator Gene of Vibrio cholerae Chromosome II
TL;DR: RctB, the replication initiator for the smaller of the two chromosomes of Vibrio cholerae, can be rate limiting for chromosome II replication, and multiple modes of control on RctB are expected to reduce fluctuations in the initiator concentration and thereby help maintain chromosome copy number homeostasis.
Journal ArticleDOI
Selective chromosome amplification in Vibrio cholerae
TL;DR: The growth rate‐insensitive behaviour of chrII, whose origin is similar to origins of members of a major class of plasmids, was shared by some but not all of several representative plasmid tested in V. cholerae, suggesting that this chromosome might serve as a repository for necessary but potentially deleterious genes.
Journal ArticleDOI
Changes in nucleoid morphology and origin localization upon inhibition or alteration of the actin homolog, MreB, of Vibrio cholerae
TL;DR: It appears that the inhibition or alteration of Vibrio MreB can affect both the nucleoid morphology and origin localization, as in mutants isolated as A22 resistant.
Journal ArticleDOI
Segregation of the replication terminus of the two Vibrio cholerae chromosomes.
TL;DR: It appears that there could be coordination between the two chromosomes through the replication and/or segregation of the terminus region to ensure their segregation to daughter cells in Vibrio cholerae.
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Transcriptional inactivation of a regulatory site for replication of Vibrio cholerae chromosome II.
TL;DR: It is found that the transcription attenuates the inhibitory activity of the gene, presumably by interfering with RctB binding, and this control circuit appears to be a putative novel mechanism for homeostasis of initiator availability.