R
R S Dykhuizen
Researcher at Aberdeen Royal Infirmary
Publications - 5
Citations - 418
R S Dykhuizen is an academic researcher from Aberdeen Royal Infirmary. The author has contributed to research in topics: Excretion & Nitrite. The author has an hindex of 5, co-authored 5 publications receiving 407 citations.
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Journal ArticleDOI
Antimicrobial effect of acidified nitrite on gut pathogens: importance of dietary nitrate in host defense.
TL;DR: Generation of salivary nitrite from dietary nitrate may provide significant protection against gut pathogens in humans, and addition of SCN-, but not that of CI-, increased the antibacterial activity.
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Protein binding and serum bactericidal activities of vancomycin and teicoplanin.
TL;DR: Despite having less in vitro cidal activity against the study isolates and having low or unrecordable levels of free drug in serum, teicoplanin demonstrated a similar or better SBA than vancomycin, which did not seem to impair its antibacterial activity as measured by its SBA.
Journal ArticleDOI
Plasma nitrate concentration in infective gastroenteritis and inflammatory bowel disease
R S Dykhuizen,J. Masson,G M McKnight,A. N. G. Mowat,Christopher Smith,Lorna Smith,Nigel Benjamin +6 more
TL;DR: In this paper, the extent of NO production in patients with infective versus non-infective forms of bowel dysfunction was compared to compare the mean and median plasma nitrate concentrations within the four groups.
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Nitric oxide synthesis in patients with infective gastroenteritis
TL;DR: Results show notably enhanced nitrate synthesis due to increased activity of thel-arginine/NO pathway in patients with infective gastroenteritis.
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The high vaginal swab in general practice: clinical correlates of possible pathogens
TL;DR: Clinical features, diagnosis and treatment of 286 women whose high vaginal swabs showed pure, heavy growth of Staphylococcus aureus, beta haemolytic streptococci groups A, C or G, Streptococcus milleri, StrePTococcus pneumoniae or Haemophilus influenzae were analysed showed that S. pneumoniae and H. Influenzae appeared to cause clinical disease.