R
Ramón Ruiz-Maldonado
Researcher at Hospital General Universitario Gregorio Marañón
Publications - 84
Citations - 2794
Ramón Ruiz-Maldonado is an academic researcher from Hospital General Universitario Gregorio Marañón. The author has contributed to research in topics: Atopic dermatitis & Urticaria pigmentosa. The author has an hindex of 29, co-authored 84 publications receiving 2660 citations. Previous affiliations of Ramón Ruiz-Maldonado include Mexican Social Security Institute.
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Giant pigmented nevi: clinical, histopathologic, and therapeutic considerations.
TL;DR: The mode of inheritance of giant pigmented nevi is probably multifactorial; four second-degree relatives of the authors' patients also had large nevi, and there was a 2:1 female predominance.
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Necrotizing fasciitis: report of 39 pediatric cases.
Antonio Fustes-Morales,Pedro Gutiérrez-Castrellón,Carola Durán-McKinster,Luz Orozco-Covarrubias,Lourdes Tamayo-Sánchez,Ramón Ruiz-Maldonado +5 more
TL;DR: Necrotizing fasciitis in children is frequently misdiagnosed, and several features differ from those of NF in adults, including immunosuppression, which was the main factor related to death.
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Keratitis, ichthyosis, and deafness (KID syndrome): review of the literature and proposal of a new terminology.
H Caceres-Rios,Lourdes Tamayo-Sánchez,Carola Durán-McKinster,M de la Luz Orozco,Ramón Ruiz-Maldonado +4 more
TL;DR: The KID acronym does not accurately define this entity since the disorder is not an ichthyosis, because scaling is not the main cutaneous feature and not all patients have keratitis early in the course, so it is suggested that this syndrome should be included under the general heading of congenital ectoderm defects as a keratodermatous ectodermal dysplasia (KED).
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Hypomelanosis of Ito: Diagnostic Criteria and Report of 41 Cases
TL;DR: A 20‐year prospective review of 41 pediatric patients with a diagnosis of hypomelanosis of Ito found no evidence pointed to hereditary transmission of the disease.
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Postinflammatory hypopigmentation and hyperpigmentation.
TL;DR: The authors propose an inherited individual chromatic tendency that is based on "weak" or "strong" melanocytes and their tendency to respond to trauma or inflammation with either hypopigmentation or hyperpigmentation.