R
Richard F. Marsh
Researcher at University of Wisconsin-Madison
Publications - 53
Citations - 3248
Richard F. Marsh is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Scrapie & Transmissible mink encephalopathy. The author has an hindex of 30, co-authored 53 publications receiving 3163 citations. Previous affiliations of Richard F. Marsh include University of California, Riverside & University of California, Davis.
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Biochemical and physical properties of the prion protein from two strains of the transmissible mink encephalopathy agent.
TL;DR: It is concluded that PrPTME from the HY and DY strains undergo different posttranslational modifications that could explain differences in the biochemical properties of PrPTme from the two sources.
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Human papovavirus (JC): induction of brain tumors in hamsters.
TL;DR: Although virus was not recovered from extracts of serially cultured tumor cells, JC virus was rescued when one tumor cell line was fused with permissive cells, and this antigen was antigenically related to SV40 T antigen.
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Identification of two biologically distinct strains of transmissible mink encephalopathy in hamsters.
TL;DR: Hamster TME passaged back into mink revealed that only DY retained mink pathogenicity, which suggests that the DY agent is the major minks pathogen in the Stetsonville TME source that is also pathogenic in hamsters after a long incubation period.
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Comparison of Scrapie and Transmissible Mink Encephalopathy in Hamsters. II. Clinical Signs, Pathology, and Pathogenesis
Richard F. Marsh,R. H. Kimberlin +1 more
TL;DR: The study of scrapie and transmissible mink encephalopathy in hamsters suggests that the cerebrospinal fluid may be an important route by which inocula are disseminated to susceptible cells after intracerebral inoculation.
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Interspecies Transmission of Chronic Wasting Disease Prions to Squirrel Monkeys (Saimiri sciureus)
TL;DR: Brain tissue from the CWD-infected squirrel monkeys contained the abnormal isoform of the prion protein, PrP-res, and displayed spongiform degeneration, the first reported transmission of CWD to primates.