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Sandra L. Watkins

Researcher at University of Washington

Publications -  53
Citations -  4020

Sandra L. Watkins is an academic researcher from University of Washington. The author has contributed to research in topics: Peritoneal dialysis & Kidney disease. The author has an hindex of 26, co-authored 52 publications receiving 3815 citations. Previous affiliations of Sandra L. Watkins include Boston Children's Hospital & Yeshiva University.

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The Risk of the Hemolytic–Uremic Syndrome after Antibiotic Treatment of Escherichia coli O157:H7 Infections

TL;DR: In this paper, a prospective cohort study of 71 children younger than 10 years of age who had diarrhea caused by E. coli O157:H7 was conducted to assess whether antibiotic treatment in these children affects the risk of the hemolytic-uremic syndrome and to assess the influence of confounding factors on this outcome.
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Escherichia coli 0157:H7 and the Hemolytic Uremic Syndrome: Importance of Early Cultures in Establishing the Etiology

TL;DR: The culture-positive group was more likely to have had bloody diarrhea and fecal leukocytes and to have received transfusions than the culture-negative group but was otherwise similar in clinical characteristics.
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Relative nephroprotection during Escherichia coli O157:H7 infections: association with intravenous volume expansion.

TL;DR: Assessment of whether pre-HUS interventions and events, especially the volume and sodium content of intravenous fluids administered early in illness, affect the risk for developing oligoanuric HUS after E coli O157:H7 infections found that these determinations probably reflect later points of these initial determinations.
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Anthropometric measures and risk of death in children with end-stage renal disease

TL;DR: In children with ESRD, growth delay and extremes in BMI are associated with an increased risk for mortality, and there was a statistically significant U-shaped association between BMI and death.
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Prothrombotic coagulation abnormalities preceding the hemolytic-uremic syndrome.

TL;DR: Thrombin generation (probably due to accelerated thrombogenesis and inhibition of fibrinolysis precede renal injury and may be the cause of such injury in the hemolytic-uremic syndrome.