S
Sang Wook Park
Researcher at Catholic University of Korea
Publications - 20
Citations - 592
Sang Wook Park is an academic researcher from Catholic University of Korea. The author has contributed to research in topics: Microsatellite instability & Cancer. The author has an hindex of 9, co-authored 20 publications receiving 531 citations.
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Journal ArticleDOI
Oncogenic NRF2 mutations in squamous cell carcinomas of oesophagus and skin.
Yoo Ri Kim,Ji Eun Oh,Min Sung Kim,Mi Ran Kang,Sang Wook Park,Ji Youn Han,Hyeon Seok Eom,Nam Jin Yoo,Sug Hyung Lee +8 more
TL;DR: The study demonstrated here that NRF2 mutation occurs not only in lung and head/neck cancers, but also in oesophageal and skin cancers, and suggests that the NRf2 mutation plays a role in the development of SCC and is a feature of S CC.
Journal ArticleDOI
Mutational and expressional analyses of ATG5, an autophagy-related gene, in gastrointestinal cancers.
TL;DR: The data suggest that ATG5 gene could be altered in gastrointestinal cancers at the mutational or expressional level and somatic mutation and loss of expression of ATG4 gene might play a role in gastrointestinal cancer pathogenesis by altering autophagic and apoptotic cell death.
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Mutational analysis of hypoxia-related genes HIF1alpha and CUL2 in common human cancers.
TL;DR: Mutational analysis of hypoxia‐related genes HIF1α and CUL2 in common human cancers in APMIS 2009 shows clear trends in prognosis for certain types of cancers.
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Mutational analysis of tumour suppressor gene NF2 in common solid cancers and acute leukaemias
TL;DR: The data suggest that somatic mutation of NF2 tumour suppressor gene may not play a central role in development of common cancers, and its mutation somatically occurs in a minor fraction of HCC, lung cancer and acute leukaemia.
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Frameshift mutations of ubiquitination-related genes HERC2, HERC3, TRIP12, UBE2Q1 and UBE4B in gastric and colorectal carcinomas with microsatellite instability
TL;DR: B-catenin immunodetection is of relative (moderate) importance and the diagnosis of desmoid-type fibromatosis can be established based on histopathological criteria and the biological significance of molecular data, particularly when they seem to be in disagreement with the histological results, must be interpreted only in relation to the clinical context.