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Shinobu Toma

Researcher at Chiba University

Publications -  16
Citations -  565

Shinobu Toma is an academic researcher from Chiba University. The author has contributed to research in topics: Microneurography & Sensory nerve. The author has an hindex of 12, co-authored 16 publications receiving 540 citations.

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Microneurography as a tool in clinical neurophysiology to investigate peripheral neural traffic in humans

TL;DR: How this technique has been used in clinical neurophysiology to elucidate the neural mechanisms of autonomic regulation, motor control and sensory functions in humans under physiological and pathological conditions is reviewed.

Invited review Microneurography as a tool in clinical neurophysiology to investigate peripheral neural traffic in humans

TL;DR: Microneurography is a method using metal microelectrodes to investigate directly identified neural traffic in myelinated and unmyelinated efferent and afferent nerves leading to and coming from muscle and skin in human peripheral nerves in situ.
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Amyotrophic cervical myelopathy in adolescence.

TL;DR: Results suggest that disproportionate shortening of the dorsal roots is further accentuated during the juvenile growth spurt, which determines the onset and self limited course of the condition, and that repeated neck flexion causes micro-trauma and relative ischaemia of anterior horn cells, which finally results in atrophy of the muscles innervated by motoneurons with long axons.
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Neuroferritinopathy in a Japanese family with a duplication in the ferritin light chain gene.

TL;DR: A 42-year-old Japanese man first developed hand tremors in his middle teens, and generalized hypotonia, hyperextensibility, aphonia, micrographia, hyperreflexia, and cognitive impairment at age 42, and his unsteady gait with long steps, with his arms and legs dangling, seemed to be due mainly to hypotonus.
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Nocturnal variation in human sympathetic baroreflex sensitivity

TL;DR: Sympathetic BRS was significantly lower during sleep than while subjects were awake in the evening, and it remained low after the subjects woke up in the morning, and this sympathetic BRS pattern may contribute to diurnal haemodynamic variables and may account, at least in part, for the connection between circadian rhythm and cardiovascular disease.