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Stefan H. Jacobson

Researcher at Karolinska Institutet

Publications -  195
Citations -  7341

Stefan H. Jacobson is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Kidney disease & Renal function. The author has an hindex of 40, co-authored 195 publications receiving 6587 citations.

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Exercise training for adults with chronic kidney disease

TL;DR: The effects of regular exercise in adults with CKD and kidney transplant patients are assessed to determine how the exercise programme should be designed to affect physical fitness and functioning, cardiovascular dimensions, nutrition, lipids, glucose metabolism, systemic inflammation, muscle morphology and morphometrics.
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Exercise Training in Adults With CKD: A Systematic Review and Meta-analysis

TL;DR: Overall, improved aerobic capacity, muscular functioning, cardiovascular function, walking capacity, and health-related quality of life were associated with various exercise interventions, although the preponderance of data were for dialysis patients and used aerobic exercise programs.
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Effect of Membrane Permeability on Survival of Hemodialysis Patients

TL;DR: The use of high-flux membranes conferred a significant survival benefit among patients with serum albumin < or = 4 g/dl, but the apparent survival Benefit among patients who have diabetes and are treated with high- flux membranes requires confirmation given the post hoc nature of the analysis.
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Histopathologic and clinical outcome of rituximab treatment in patients with cyclophosphamide-resistant proliferative lupus nephritis.

TL;DR: For patients with proliferative lupus nephritis who fail to respond to conventional immunosuppressive therapy including CYC, combined treatment with rituximab and CYC may constitute a new treatment option.
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Prenatal Dexamethasone Causes Oligonephronia, Sodium Retention, and Higher Blood Pressure in the Offspring

TL;DR: It is found that high levels of maternal glucocorticoids impair renal development and lead to arterial hypertension in offspring, and even though renal mass eventually normalizes, glomerular damage as well as sodium retention occur and these factors may contribute to the development of hypertension.