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Stephan Martin

Researcher at University of Düsseldorf

Publications -  142
Citations -  6114

Stephan Martin is an academic researcher from University of Düsseldorf. The author has contributed to research in topics: Diabetes mellitus & Type 2 diabetes. The author has an hindex of 41, co-authored 139 publications receiving 5424 citations. Previous affiliations of Stephan Martin include Leibniz Association.

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Environmental/lifestyle factors in the pathogenesis and prevention of type 2 diabetes

TL;DR: A comprehensive overview of factors contributing to diabetes risk, including aspects of diet quality and quantity, little physical activity, increased monitor viewing time or sitting in general, exposure to noise or fine dust, short or disturbed sleep, smoking, stress and depression, and a low socioeconomic status are presented.
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Effects of coffee consumption on subclinical inflammation and other risk factors for type 2 diabetes: a clinical trial

TL;DR: Coffee consumption appears to have beneficial effects on subclinical inflammation and HDL cholesterol, whereas no changes in glucose metabolism were found in the authors' study.
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Impaired glucose tolerance is associated with increased serum concentrations of interleukin 6 and co-regulated acute-phase proteins but not TNF-α or its receptors

TL;DR: This study shows systemic up-regulation of selected inflammatory mediators in patients with Type II diabetes and IGT and the pattern observed is non-random and fits with an IL-6 associated rather than TNF-α associated response.
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Self-monitoring of blood glucose in type 2 diabetes and long-term outcome: an epidemiological cohort study.

TL;DR: SMBG was associated with decreased diabetes-related morbidity and all-cause mortality in type 2 diabetes, and this association remained in a subgroup of patients who were not receiving insulin therapy, and may be associated with a healthier lifestyle and/or better disease management.
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Development of type 1 diabetes despite severe hereditary B-cell deficiency.

TL;DR: The role of B cells and autoantibodies in type 1 diabetes in humans has not been fully resolved, but T cells that are reactive to several islet autoantigens have been identified in both mice and humans.