S
Stephen J. Winters
Researcher at University of Pittsburgh
Publications - 93
Citations - 3320
Stephen J. Winters is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Gonadotropin & Gonadotropin secretion. The author has an hindex of 34, co-authored 87 publications receiving 3261 citations. Previous affiliations of Stephen J. Winters include National Institutes of Health & University of Louisville.
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Endocrine consequences of weight loss in obese, hyperandrogenic, anovulatory women *†
TL;DR: Weight loss in obese, hyperandrogenic, anovulatory women appears to reduce insulin and non-SHBG T concentrations despite the absence of a change in gonadotropin secretion and may lead to resumption of ovulation as mentioned in this paper.
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Visceral obesity and insulin resistance are associated with plasma aldosterone levels in women.
TL;DR: It is concluded that visceral adiposity and insulin resistance are associated with increased plasma aldosterone and other adrenal steroids that may contribute to cardiovascular diseases in obese women.
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The analog free testosterone assay: are the results in men clinically useful?
TL;DR: The Coat-A-Count free testosterone concentration in men, like the total testosterone concentration, is determined in part by plasma SHBG, and androgen deficiency may be misclassified with this assay in men with low SHBG.
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The gonadotropin-suppressive activity of androgen is increased in elderly men
Stephen J. Winters,Stephen J. Winters,Richard J. Sherins,Richard J. Sherins,Philip Troen,Philip Troen +5 more
TL;DR: The data indicate that elderly men are more responsive than are young men to the gonadotropin-suppressive effects of androgen, but not to DHT effects on circulating TeBG levels.
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Episodic Luteinizing Hormone (LH) Secretion and the Response of LH and Follicle-Stimulating Hormone to LH-Releasing Hormone in Aged Men: Evidence for Coexistent Primary Testicular Insufficiency and an Impairment in Gonadotropin Secretion*
Stephen J. Winters,Philip Troen +1 more
TL;DR: The data indicate that Leydig cell function is impaired in healthy elderly men as a result of primary testicular insufficiency and reveal the presence of an additional hypothalamic-pituitary disorder of gonadotropin secretion associated with the aging process.