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Tami Katzir

Researcher at University of Haifa

Publications -  74
Citations -  1666

Tami Katzir is an academic researcher from University of Haifa. The author has contributed to research in topics: Reading (process) & Reading comprehension. The author has an hindex of 22, co-authored 58 publications receiving 1394 citations. Previous affiliations of Tami Katzir include University of California, Los Angeles & Harvard University.

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Reading fluency: the whole is more than the parts.

TL;DR: The results demonstrate that phonological awareness, rapid letter naming, and orthographic pattern recognition contribute to word-reading skills and support the multidimensional nature of fluency in which the whole is more than its parts.
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The Role of Reading Self-Concept and Home Literacy Practices in Fourth Grade Reading Comprehension.

TL;DR: This article examined the relationships among reading comprehension, reading self-concept, and home literacy environment (HLE) in a sample (n = 67) of fourth grade children enrolled in an urban school district.
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The Varieties of Pathways to Dysfluent Reading Comparing Subtypes of Children With Dyslexia at Letter, Word, and Connected Text Levels of Reading

TL;DR: The results demonstrated that the three DDH subtypes exhibited differences in fluency at different levels of reading (letter, word, and connected text), underscoring the separate reading profiles of these subtypes and the different possible routes to dysfluency in reading disabilities.
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fMRI of syntactic processing in typically developing children: structural correlates in the inferior frontal gyrus.

TL;DR: Relationships between structural and functional neurodevelopment that co-occur with improved syntactic processing in critical language regions of the IFG in typically developing children are shown.
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A structural basis for reading fluency: white matter defects in a genetic brain malformation.

TL;DR: It is demonstrated that a genetic disorder of gray matter heterotopia shares behavioral characteristics with developmental dyslexia, and that focal white matter defects in this disorder may serve as the structural brain basis of this phenomenon.