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Teresa M. Evans

Researcher at University of Texas Health Science Center at San Antonio

Publications -  17
Citations -  814

Teresa M. Evans is an academic researcher from University of Texas Health Science Center at San Antonio. The author has contributed to research in topics: Mental health & Mentorship. The author has an hindex of 7, co-authored 17 publications receiving 474 citations. Previous affiliations of Teresa M. Evans include University of Cincinnati & University of Texas at Austin.

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Evidence for a mental health crisis in graduate education

TL;DR: With mental illness a growing concern within graduate education, data from a new survey should prompt both academia and policy makers to consider intervention strategies.
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Rapamycin Treatment Improves Neuron Viability in an In Vitro Model of Stroke

TL;DR: It is shown that rapamycin can prevent the activation of both mTORC1 and m TORC2 in cortical neurons and improve cell survival following oxygen glucose deprivation, an in vitro model of ischemic stroke.
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Microwave and magnetic (M2) proteomics of a mouse model of mild traumatic brain injury

TL;DR: The proof-of-concept that M2 proteomics is a rapid method to quantify putative protein biomarkers and therapeutic targets of mTBI is supported and the feasibility of CSP expression correlations to long-term effects on motor impairment is suggested.
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Mental Health Crisis in Graduate Education: The Data and Intervention Strategies

TL;DR: There is a growing cry for help from graduate students and postdoctoral fellows (trainees) across the globe who struggle with significant mental health concerns as mentioned in this paper, however, although there has been increased d...
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The effect of mild traumatic brain injury on peripheral nervous system pathology in wild-type mice and the G93A mutant mouse model of motor neuron disease.

TL;DR: It is demonstrated that mild-TBI induces inflammation and oxidative stress and negatively impacts muscle denervation and motor performance, suggesting mild- TBI can potentiate motor neuron pathology and influence the development of MND in mice.