Teresa Wierzba-Bobrowicz

TL;DR: A pivotal role of microglial cells in repair processes and/or etiopathogenesis of schizophrenia is suggested and indicates that they undergo substantial damage in the course of chronic schizophrenia.

TL;DR: The results indicate that sprouting angiogenesis is a quantitatively significant source of vessels in the cerebral infarct border zone, however, non-therapeutically stimulated angiogenic developed only 3-4 days after the stroke, that is beyond the period of reversible changes in ischemic penumbra recognized as a "therapeutical window" in the human brain.

TL;DR: Treatment of chronic schizophrenics should involve the supply of agents to prevent degeneration of microglia and/or long-term immunotherapy, and morphological signs of the former normal function of immunocompetent and phagocytosing cells.

TL;DR: Comparisons between the cases of Parkinson's disease with dementia (PDD) and those without (PD-only) revealed that the former were characterised by significantly higher densities of LBs and LNs as well as in the CA2-3 region of the hippocampus and cortical complex of amygdala, suggesting the spread of the pathological process via axonal transport.

TL;DR: In the present study immunohistochemical identification of rod microglial phenotype against ramified microglia was performed on segments of 17 brains derived from 7 cases of encephalitis of viral aetiology, 6 cases of Wilson's disease and 4 cases of Alzheimer's disease, revealing a strong similarity between immunohISTochemical phenotypes of both morphological forms ofmicroglia.