T
Thomas Müller
Researcher at Ruhr University Bochum
Publications - 371
Citations - 20977
Thomas Müller is an academic researcher from Ruhr University Bochum. The author has contributed to research in topics: Catalysis & Parkinson's disease. The author has an hindex of 58, co-authored 360 publications receiving 18967 citations. Previous affiliations of Thomas Müller include Bayer & Bayer MaterialScience.
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Hydroamination on homogeneous and heterogeneous catalysts: Kinetic study
TL;DR: In this article, the authors compared homogeneous and heterogeneous catalysts for the cyclization of 6-aminohex-1-yne and showed that the metal catalyzed hydroamination reaction is followed by a proton-catalyzed isomerization of the primary reaction product, 2-methylene-piperidine.
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Modulation of disease risk according to a cathepsin D / apolipoprotein E genotype in Parkinson's disease.
Thorsten Schulte,Stefan Böhringer,Ludger Schöls,Thomas Müller,C. Fischer,Olaf Riess,Horst Przuntek,Klaus Berger,Jörg T. Epplen,Rejko Krüger +9 more
TL;DR: Interference of CTSD and APOE polymorphisms in the pathogenesis of PD, in the sense of modulating disease risk, is suggested.
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Prevalence and treatment strategies of dyskinesia in patients with Parkinson's disease.
TL;DR: A study into the prevalence and treatment of dyskinesia in Parkinson’s disease patients was performed with 380 PD specialists-completed interviews relating to PD and retrospectively completed 1900 patient record forms for patients with dysKinesia.
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CO 2 Chemistry
Thomas Müller,Walter Leitner +1 more
TL;DR: This Thematic Series on CO2 chemistry for the Beilstein Journal of Organic Chemistry (BJOC) highlights the growing demand for energy, materials and chemicals and the need for more resource-efficient chemical processes.
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Comparison of 200 mg retarded release levodopa/carbidopa - with 150 mg levodopa/carbidopa/entacapone application: pharmacokinetics and efficacy in patients with Parkinson's disease.
TL;DR: LD/CD/EN was superior to LD/CD regarding the attention related components of line tracing probably due to a hypothetically increased dopamine occurrence at the prefrontal cortex, which guides human behaviour.