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Udo Meyerfeldt

Researcher at Humboldt University of Berlin

Publications -  15
Citations -  1581

Udo Meyerfeldt is an academic researcher from Humboldt University of Berlin. The author has contributed to research in topics: Heart rate variability & Ventricular tachycardia. The author has an hindex of 10, co-authored 15 publications receiving 1441 citations. Previous affiliations of Udo Meyerfeldt include Humboldt State University.

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Recurrence-plot-based measures of complexity and their application to heart-rate-variability data.

TL;DR: Applying measures of complexity based on vertical structures in recurrence plots and applying them to the logistic map as well as to heart-rate-variability data is able to detect and quantify the laminar phases before a life-threatening cardiac arrhythmia occurs thereby facilitating a prediction of such an event.
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Nonlinear analysis of complex phenomena in cardiological data

TL;DR: It is demonstrated that parameters from nonlinear dynamics are useful for risk stratification after myocardial infarction, for the prediction of life-threatening cardiac events even in short time series, and for modelling the relationship between heart rate and blood pressure regulation.
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Short-term forecasting of life-threatening cardiac arrhythmias based on symbolic dynamics and finite-time growth rates.

TL;DR: It is found that neither the time nor the frequency domain parameters show significant differences between the VT-VF and the control time series, however, two parameters from symbolic dynamics as well as the finite-time growth rates discriminate significantly both groups.
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Forecasting of life threatening arrhythmias using the compression entropy of heart rate.

TL;DR: Compression entropy might be a suitable parameter for short-term forecasting of life-threatening tachycardia in ICD.
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Heart rate variability before the onset of ventricular tachycardia: differences between slow and fast arrhythmias.

TL;DR: It is found that the onset of slow VT was characterized by a significant increase in heart rate, whereas fast VT was triggered during decreased heart rates, compared to the control series.