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Wendy A. Henderson

Researcher at University of Connecticut

Publications -  95
Citations -  1861

Wendy A. Henderson is an academic researcher from University of Connecticut. The author has contributed to research in topics: Irritable bowel syndrome & Microbiome. The author has an hindex of 21, co-authored 87 publications receiving 1283 citations. Previous affiliations of Wendy A. Henderson include University of Pittsburgh & Boston Children's Hospital.

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Gut Microbiome Developmental Patterns in Early Life of Preterm Infants: Impacts of Feeding and Gender.

TL;DR: Investigating day-to-day gut microbiome patterns in preterm infants during their first 30 days of life in the neonatal intensive care unit (NICU) found that infant postnatal age, gender and feeding type significantly contribute to the dynamic development of the gut microbiome in pre term infants.
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Altered Composition of Gut Microbiota in Depression: A Systematic Review.

TL;DR: Alteration of gut microbiome patterns was evident in people with depression and further evidence is warranted to allow for the translation of microbiome findings toward innovative clinical strategies that may improve treatment outcomes inPeople with depression.
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Dose response of PEG 3350 for the treatment of childhood fecal impaction

TL;DR: In this paper, the efficacy and safety of polyethylene glycol (PEG) 3350 in the treatment of childhood colorectal impaction was investigated, using a prospective, double-blind, parallel, randomized study.
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The impact of cumulative pain/stress on neurobehavioral development of preterm infants in the NICU.

TL;DR: Understanding mechanisms by which early life experience alters neurodevelopment will assist clinicians in developing targeted neuroprotective strategies and individualized interventions to improve infant developmental outcomes.
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Elevated circulating miR-150 and miR-342-3p in patients with irritable bowel syndrome.

TL;DR: This preliminary study reports the association of two miRNAs, detected in whole blood, with IBS, which link to pain and inflammatory pathways both of which are thought to be dysregulated in IBS.