W
William J. Koch
Researcher at University of British Columbia
Publications - 26
Citations - 2630
William J. Koch is an academic researcher from University of British Columbia. The author has contributed to research in topics: Anxiety disorder & Panic disorder. The author has an hindex of 18, co-authored 26 publications receiving 2567 citations.
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Journal ArticleDOI
How does anxiety sensitivity vary across the anxiety disorders
TL;DR: In this article, a measure of anxiety sensitivity known as the Anxiety Sensitivity Index (ASI) was used to assess 313 patients, representing each of the six DSM-III-R anxiety disorders.
Journal ArticleDOI
Cognitive versus behavior therapy in the group treatment of obsessive-compulsive disorder.
Peter D. McLean,Maureen L. Whittal,Dana S. Thordarson,Steven Taylor,Ingrid Söchting,William J. Koch,Randy J. Paterson,Kent W. Anderson +7 more
TL;DR: In terms of clinically significant improvement, treatment groups were equivalent on the conclusion of treatment, but 3 months later significantly more ERP participants met criteria for recovered status, and the extent of this cognitive change was similar between CBT and ERP groups.
Journal ArticleDOI
Anxiety sensitivity and depression: how are they related?
TL;DR: In this paper, the authors investigated the relationship between anxiety sensitivity and depression and revealed three factors of anxiety sensitivity: fear of publicly observable symptoms, fear of loss of cognitive control, and fear of bodily sensations.
Journal ArticleDOI
Posttraumatic stress disorder arising after road traffic collisions: Patterns of response to cognitive-behavior therapy.
Steven Taylor,Ingrid C. Fedoroff,William J. Koch,Dana S. Thordarson,Gary Fecteau,Richard M. Nicki +5 more
TL;DR: Two preliminary studies suggest that cognitive-behavior therapy (CBT) is, on average, effective in treating posttraumatic stress disorder, although the major patterns of treatment outcome remain to be identified.
Journal ArticleDOI
The structure of posttraumatic stress symptoms.
TL;DR: The results suggest that posttraumatic stress reactions arise from a general mechanism, with contributions from 2 specific mechanisms.