W
Wolfgang Leibold
Researcher at Cedars-Sinai Medical Center
Publications - 28
Citations - 1165
Wolfgang Leibold is an academic researcher from Cedars-Sinai Medical Center. The author has contributed to research in topics: Epstein–Barr virus & Antigen. The author has an hindex of 13, co-authored 28 publications receiving 1161 citations. Previous affiliations of Wolfgang Leibold include Karolinska Institutet & University of California, Los Angeles.
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Journal Article
Establishment and characterization of an Epstein-Barr virus (EBC)-negative lymphoblastoid B cell line (BJA-B) from an exceptional, EBV-genome-negative African Burkitt's lymphoma
TL;DR: BJA-B is the firstEBV-negative LCL established from an African Burkitt's lymphoma and demonstrates that an EBV-independent continuous B cell line can be established "in vitro" from other than leukemia or myeloma cells.
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Continuous Lymphoid Cell Lines with Characteristics of B Cells (Bone-Marrow-Derived), Lacking the Epstein-Barr Virus Genome and Derived from Three Human Lymphomas
George Klein,Tomas L. Lindahl,Mikael Jondal,Wolfgang Leibold,José Menezes,Kenneth Nilsson,Christer Sundström +6 more
TL;DR: Three exceptional cell lines have been tested for the presence of the Epstein-Barr virus genome by nucleic acid hybridization (complementary RNA) and virus-determined nuclear antigen tests and two clearly had characteristics of B-cells (bone-marrow-derived).
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Biological differences between Epstein-Barr virus (EBV) strains with regard to lymphocyte transforming ability, superinfection and antigen induction.
TL;DR: Transformation of CBL as contrasted to EA induction in, and growth inhibition of Raji cells thus appear as mutually exclusive viral functions.
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Infection of EBV-Genome-Negative and – Positive Human Lymphoblastoid Cell Lines with Biologically Different Preparations of EBV
TL;DR: Findings show that different EBV isolates may differ in their biological activity, probably due to their having different degrees of viral dependence on restrictive host cell controls.
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HLA-D locus typing in ankylosing spondylitis and Reiter's syndrome.
Kip Kemple,Richard A. Gatti,Richard A. Gatti,Wolfgang Leibold,Wolfgang Leibold,James R. Klinenberg,James R. Klinenberg,Rodney Bluestone +7 more
TL;DR: HLA-D typing of patients with ankylosing spondylitis and Reiter's syndrome did not show increased frequency of any particular Dw allele in either population of patients as compared to controls, and hyperresponsiveness in both AS and RS patients was found.