Y
Yasufumi Omori
Researcher at Akita University
Publications - 47
Citations - 1328
Yasufumi Omori is an academic researcher from Akita University. The author has contributed to research in topics: Connexin & Head and neck squamous-cell carcinoma. The author has an hindex of 21, co-authored 39 publications receiving 1241 citations.
Papers
More filters
Journal Article
Alterations of (CA)n DNA Repeats and Tumor Suppressor Genes in Human Gastric Cancer
Nikolai Mironov,M A Aguelon,G.I. Potapova,Yasufumi Omori,O.V. Gorbunov,A.A. Klimenkov,Hiroshi Yamasaki +6 more
TL;DR: The results suggest that genomic instability revealed by (CA)n repeat changes does not seem to contribute to induction of point mutations in p53 or connexin 32 genes but may participate in loss of heterozygosity at APC/MCC loci.
Journal ArticleDOI
Downregulation of gap junctions in cancer cells.
TL;DR: Current knowledge on the molecular mechanisms involved in the downregulation of gap junctions in cancer cells is reviewed to discuss the potential of connexins as targets in cancer prevention and therapy.
Journal ArticleDOI
Transdifferentiation of Mature Rat Hepatocytes into Bile Duct-Like Cells in Vitro
Yuji Nishikawa,Yuko Doi,Hitoshi Watanabe,Takuo Tokairin,Yasufumi Omori,Mu Su,Toshiaki Yoshioka,Katsuhiko Enomoto +7 more
TL;DR: The results suggest that hepatocytes might have the potential to transdifferentiate into bile duct-like cells without acquiring a stem-like phenotype and that this is mediated through specific protein tyrosine phosphorylation pathways.
Journal ArticleDOI
Cell biology and pathology of liver sinusoidal endothelial cells.
Katsuhiko Enomoto,Yuji Nishikawa,Yasufumi Omori,Takuo Tokairin,Masayuki Yoshida,Naoto Ohi,Takuya Nishimura,Youhei Yamamoto,Qinchang Li +8 more
TL;DR: It is suggested that SEC, cooperating with Kupffer cells and hepatic dendritic cells, may partake of immunoregulatory functions in the liver, and the mechanisms of apoptosis and proliferation of SEC are elucidated.
Journal ArticleDOI
Connexin26‐mediated gap junctional communication reverses the malignant phenotype of MCF‐7 breast cancer cells
Megumi Momiyama,Yasufumi Omori,Yasuko Ishizaki,Yuji Nishikawa,Takuo Tokairin,Junichi Ogawa,Katsuhiko Enomoto +6 more
TL;DR: The results strongly suggest that the induction of Cx 26 protein observed in human breast cancers may not be very relevant to the development of breast cancers, and that Cx26 can function as a tumor suppressor in breast cancer cells.