Y
Yi Liu
Researcher at Huazhong University of Science and Technology
Publications - 59
Citations - 1301
Yi Liu is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Stromal cell & Cell migration. The author has an hindex of 17, co-authored 52 publications receiving 828 citations.
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Quaternized chitosan-Matrigel-polyacrylamide hydrogels as wound dressing for wound repair and regeneration
Hang Xue,Liangcong Hu,Yuan Xiong,Xiangwei Zhu,Congying Wei,Faqi Cao,Wu Zhou,Yun Sun,Yori Endo,Mengfei Liu,Yi Liu,Jing Liu,Abudula Abududilibaier,Lang Chen,Chenchen Yan,Bobin Mi,Guohui Liu +16 more
TL;DR: The results of histology and molecular testing in vivo demonstrated that the hybrid hydrogel could significantly enhance wound healing, collagen deposition, and induce skin adnexal regeneration by upregulating anti-inflammatory factors, and downregulating proinflammatory factors.
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Hypoxia-inducible factor-1α promotes endometrial stromal cells migration and invasion by upregulating autophagy in endometriosis.
TL;DR: Results suggest that HIF-1α promotes HESCs invasion and metastasis by upregulating autophagy, and inhibition of autophileagy might be a novel therapeutic approach to the treatment of endometriosis.
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Hypoxia-inducible factor 1α-induced epithelial–mesenchymal transition of endometrial epithelial cells may contribute to the development of endometriosis
TL;DR: Overexpression of Hif-1α and changes associated with EMT were observed in normal, eutopic and ectopic endometrial tissues and hypoxia-stabilized HIF-1 α may play an important role in the invasion of endometriosis.
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Icariin Activates Autophagy via Down-Regulation of the NF-κB Signaling-Mediated Apoptosis in Chondrocytes.
TL;DR: Results indicated that icariin protected against OA by suppressing inflammatory cytokines and apoptosis, through activation of autophagy via NF-κB inhibition, making it a promising compound for cartilage tissue engineering in the treatment of OA.
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17 β-Estradiol promotes vascular endothelial growth factor expression via the Wnt/β-catenin pathway during the pathogenesis of endometriosis.
TL;DR: Promising results improve the body of knowledge on endometriosis pathogenesis and could open up new therapeutic strategies for the treatment of endometRIosis.