Z
Zahra Ghanian
Researcher at University of Wisconsin–Milwaukee
Publications - 19
Citations - 126
Zahra Ghanian is an academic researcher from University of Wisconsin–Milwaukee. The author has contributed to research in topics: Oxidative stress & Retinal. The author has an hindex of 7, co-authored 18 publications receiving 116 citations. Previous affiliations of Zahra Ghanian include Amirkabir University of Technology & Food and Drug Administration.
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Journal ArticleDOI
Optical imaging of mitochondrial redox state in rodent model of retinitis pigmentosa
Sepideh Maleki,Sandeep Gopalakrishnan,Zahra Ghanian,Reyhaneh Sepehr,Heather Schmitt,Janis T. Eells,Mahsa Ranji +6 more
TL;DR: Cryo-fluorescence redox imaging was used as a quantitative marker of OS in eyes from transgenic rats and demonstrated that alterations in the oxidative state of eyes occur during the early stages of RP.
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Effects of p67phox on the mitochondrial oxidative state in the kidney of Dahl salt-sensitive rats: optical fluorescence 3-D cryoimaging.
Fahimeh Salehpour,Zahra Ghanian,Chun Yang,Nadezhda N. Zheleznova,Theresa Kurth,Ranjan K. Dash,Allen W. Cowley,Mahsa Ranji +7 more
TL;DR: It is concluded that the p67(phox) subunit of NADPH oxidase 2 plays an important role in the excess production of ROS from mitochondria in the renal medulla of the SS rat.
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Optical imaging of mitochondrial redox state in rodent models with 3-iodothyronamine.
Zahra Ghanian,Sepideh Maleki,Hannah Reiland,Daniel E. Butz,Grazia Chiellini,Fariba-Assadi Porter,Mahsa Ranji +6 more
TL;DR: While T1AM at a high dose increases oxidative response in kidneys, it has a protective effect in the heart and may exert its effect through alternative pathways at different doses and at tissue specific levels.
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Quantitative optical measurement of mitochondrial superoxide dynamics in pulmonary artery endothelial cells
TL;DR: The results showed that mitochondrial respiratory complexes I, III and IV are sources of O2•- production in PAECs, and a new observation that ROS production during uncoupling of mitochondrial respiration is mediated in part via complex IV.
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Organ specific optical imaging of mitochondrial redox state in a rodent model of hereditary hemorrhagic telangiectasia-1.
TL;DR: Endoglin haploinsufficiency is associated with less oxidative stress in various organs and mitigation of angiogenesis in mice model of HHT‐1.