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Zongxin Ling

Researcher at Zhejiang University

Publications -  49
Citations -  4971

Zongxin Ling is an academic researcher from Zhejiang University. The author has contributed to research in topics: Dysbiosis & Gut flora. The author has an hindex of 22, co-authored 47 publications receiving 3214 citations. Previous affiliations of Zongxin Ling include Linyi University.

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Altered fecal microbiota composition in patients with major depressive disorder

TL;DR: Fecal samples from 46 patients with depression are analyzed to enable a better understanding of changes in the fecal microbiota composition in such patients, showing either a predominance of some potentially harmful bacterial groups or a reduction in beneficial bacterial genera.
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The Human Microbiota in Health and Disease

TL;DR: This paper focuses on the interactions between the human microbiota and the host in order to provide an overview of the microbial role in basic biological processes and in the development and progression of major human diseases such as infectious diseases, liver diseases, gastrointestinal cancers, metabolic diseases, respiratory diseases, mental or psychological diseases, and autoimmune diseases.
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Molecular analysis of the diversity of vaginal microbiota associated with bacterial vaginosis

TL;DR: The data presented here have clearly profiled the overall structure of vaginal communities and clearly demonstrated that BV is associated with a dramatic increase in the taxonomic richness and diversity of vaginal microbiota.
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Altered Fecal Microbiota Composition Associated with Food Allergy in Infants

TL;DR: Detailed microbiota analysis of a well-characterized cohort of infants with FA showed that dysbiosis of fecal microbiota with several FA-associated key phylotypes may play a pathogenic role in FA.
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Maternal infection during pregnancy and risk of autism spectrum disorders: A systematic review and meta-analysis.

TL;DR: It is found that maternal infection during pregnancy increases the risk of ASD in offspring, and possible mechanisms may include direct effects of pathogens and, more indirectly, the effects of inflammatory responses on the developing brain.