Showing papers by "Hebron University published in 1995"
TL;DR: A hypothesis is developed that explains the physiopathologic mechanism that causes the clinical changes described in each case of patients with papillary colobomatous defects, bearing in mind the embryologic origin and histologic structure of these anomalies.
Abstract: We describe congenital colobomatous malformations of the optic nerve and the clinical variations and complications that may appear eventually in them. We also present two clinical cases of patients with papillary colobomatous defects where spontaneous variations have been observed throughout their evolution and where the current ophthalmoscopic aspect is completely different from the initial one. Finally, bearing in mind the embryologic origin and histologic structure of these anomalies, we develop a hypothesis that explains the physiopathologic mechanism that causes the clinical changes described in each case.
9 citations
01 Jan 1995
TL;DR: In this article, relative changes in AVDO2 after manipulations of mean arterial blood pressure and arterial pCO2 were used to assess CO2 reactivity and autoregulation in severe head injury patients.
Abstract: It has been demonstrated both clinically and in experimental models that autoregulation and CO2 reactivity can be impaired independently of each other in many brain insults, the so-called dissociated vasoparalysis [1]. The theoretical combination of preserved CO2 reactivity and impaired or absent autoregulation can have many clinical implications in the overall daily management of brain-injured patients. To optimize their treatment, a bedside assessment of autoregulation and CO2 reactivity is desirable. In spite of some unresolved and controversial methodological problems, monitoring hemodynamic parameters through a reverse catheter with its tip in the jugular bulb is an easy way of monitoring brain metabolism and cerebral blood flow (CBF) coupling and in some cases of estimating CBF [2-4]. When the cerebral metabolic rate of oxygen (CMRO2) is constant, changes in arteriojugular differences of oxygen (AVDO2) reflect changes in CBF [5]. In this situation, relative changes in AVDO2 can be viewed as inverse changes in CBF and used as an evaluation method of CO2 reactivity and autoregulation. Our aims in this chapter are to use relative changes in AVDO2 after manipulations of mean arterial blood pressure and arterial pCO2 to assess CO2 reactivity and autoregulation in severe head injury patients.
6 citations
TL;DR: An unsuspected giant fusiform aneurysm arising from the intracavernous segment of the internal carotid artery associated with a growth-hormone secreting pituitary adenoma is presented and contrast-enhanced MRA permitted a better depiction of its relation with the carotids.
Abstract: An unsuspected giant fusiform aneurysm arising from the intracavernous segment of the internal carotid artery associated with a growth-hormone secreting pituitary adenoma is presented. The aneurysm was diagnosed with MRI using conventional spin-echo sequences, however, contrast-enhanced MRA permitted a better depiction of its relation with the carotid artery. The MR angiogtaphy technique may add useful information to conventional MRI, in the preoperative assessment of pituitary tumours.
1 citations
TL;DR: PSA levels were analyzed in 61 men with chronic renal failure and they did not find any significant changes related to hemodialysis, but Monath et al (2) found a small but significant increase in post-dialysis PSA levels in a group of 26 patients.
Abstract: Sirs, PSA serum levels are firstly associated with the amount of prostatic epithelial cells producing this protein and the degree of its production, secondly with the elimination factors to the circulation, and thirdly with its catabolism in serum or elsewhere in the body. The role of the kidney and liver is still unknown. In order to assess the impact of the metabolism of both organs on PSA serum levels we have measured this antigen in the serum of 178 healthy male blood donors as a control group (CG), in 48 males with chronic liver failure due to cirrhosis (LFG) and in 48 males with chronic renal failure (RFG) , 22 of whom in a hemodialysis program in which we have determined pre and post-dialysis PSA serum levels. Quantification of PSA was performed by an IRMA PSA (Tandem, Hybritech) in sera stored at -20°e. The mean age was 41.9 years (20-68) in the CG, 48.5 (16-76) in the RFG and 52.7 (29-69) in the LFG. The mean PSA serum level was 1.37 ng/ml (0.2-6.2) in the CG, 1.33 ng/ml (0.2-6.1) in the RFG and 0.62 ng/ml (0.2-4) in the LFG. A significant decrease in the mean PSA level (p