Showing papers on "Anterior accessory saphenous vein published in 2013"
TL;DR: A 56-year-old male with swelling in left inguinal region and symptoms of chronic venous insufficiency is presented with an aneurysm in left accessory saphenous vein and resected under local anesthesia.
Abstract: Venous aneurysms are rare entities observed in neck, thorax, extremity, and abdominal veins. Although the exact etiology is unknown, trauma to vessel wall, inflammation, congenital anomalies, and local degenerative changes are implicated. Venous aneurysms may be thrombosed and lead to thromboembolic events, acute pulmonary embolism, and death. Resection of the aneurysm is the general preferred approach. We present a 56-year-old male with swelling in left inguinal region and symptoms of chronic venous insufficiency. Diag - nostic workup revealed an aneurysm in left accessory saphenous vein. It was resected under local anesthesia. The patient was discharged without any complications.
1 citations
TL;DR: The current study showed that the segmental aplasia of the GSV was seen in one-third of limbs on each side and was mostly unilateral; it was always present in its mid portion below or above the knee.
Abstract: This is a prospective study to assess the frequency and anatomic distribution of the segmental absence or aplasia of the GSV using ultrasonography. 670 limbs of 335 consecutive patients who had signs and symptoms related to venous insufficiency of the leg were evaluated. Venous clinical severity scores ranged from 0 to 20The GSV was examined for its diameter, its relation with the fascial compartments and venous reflux on both legs. Diagnosis of segmental absence of the GSV was established when ultrasonography showed that the saphenous vein left the compartment and there was not any other saphenous vein in it. If a normal diameter or smaller than normal diameter vein remained in the compartment all along its course, this was not considered segmental aplasia and excluded from the study. Segmental aplasia was classified into three subgroups. Type 1: The GSV leaves the saphenous compartment in the leg and joins it at any point in the thigh. Type 2: The GSV leaves the saphenous compartment in the leg and joins it in the leg just below the knee. Type 3: The GSV leaves the saphenous compartment in the thigh and joins it more cranially in the thigh. The current study showed that the segmental aplasia of the GSV was seen in one-third of limbs on each side and was mostly unilateral; it was always present in its mid portion below or above the knee . It was found in 223 of 670 limbs (33%) in the whole patient population. It was type 1 in 59%, type 2 in 29%, and type 3 in 12% of the patients. It was was seen in 65 of 189 limbs (34.4%) with GSV insufficiency and 45 of 146 limbs (30.8%) with normal GSV on the right side, and 65 of 194 limbs (33.5%) with GSV insufficiency and 44 of 141 limbs (31.2%) with normal GSV on the left side. There was no relation between the presence of segmental aplasia of the GSV and the presence of GSV or SSV insufficiency in the same limb among patients with CEAP scores 1 and above. Aplasic segment of the GSV may prevent progression of any kind of endovenous device such as surgical stripper, laser fiber, or radiofrequency ablation probe. Care must be taken not to cause thermal damage during endovenous thermal ablation of the insufficient connecting or bridging vein as this vein comes closer to the skin after leaving the saphenous compartment. This vein was named as accessory saphenous vein or tributary vein. An alternative term such as the saphenous connecting vein or bridging vein regarding its function or the saphenous bow regarding its shape might be more appropriate. The etiology of segmental aplasia or hypoplasia of the GSV is unknown. It was assumed to be due to a developmental prevalence of vessels with the most favorable hemodynamic condition over the greater vessels that underwent atrophy.
1 citations
01 Jan 2013
TL;DR: A 55-year-old male patient, who had a transient ischaemic attack, lasting 30 minutes, after foam sclerotherapy of the small saphenous vein, is assumed to have had the cause of the TIA, even though a cardiac cause cannot be ruled out with absolute certainty.
Abstract: Summary We report on the case of a 55-year-old male patient, who had a transient ischaemic attack (TIA), lasting 30 minutes, after foam sclerotherapy of the small saphenous vein. The patient had marked small saphenous varicose veins on both sides as well as varicosities of the anterior accessory saphenous vein in the left leg. CEAP classification was stage 3 on both sides. The patient had a history of chronic atrial fibrillation with severe sick sinus syndrome and was on therapeutic anticoagulation. He also had dilated cardiomyopathy with an ejection fraction of only 35 %. Following two unremarkable sessions of foam sclerotherapy with 0.5 % foamed polidocanol applied to the anterior accessory saphenous vein, the patient had a transient ischaemic attack shortly after completion of the third session, in which the small saphenous vein was treated, likewise with foam sclerotherapy with 0.5 % foamed polidocanol. He experienced weakness of the left leg, reduced strength in the left hand and numbness in both limbs on the left side. The symptoms resolved completely within 30 minutes. A cardiology work-up prior to the intervention excluded the presence of a patent foramen ovale or intracardiac thrombi. Carotid artery stenosis was ruled out as the cause of the TIA by colour duplex ultrasonography. As ECG monitoring after the occurrence of the TIA showed that the heart rhythm remained stable, without any bradycardia or long pauses, we assume that the foam sclerotherapy was the cause of the TIA, even though a cardiac cause cannot be ruled out with absolute certainty.