Showing papers on "Intracranial Lipoma published in 1995"

Lipoma in the quadrigeminal cistern--case report.

Abstract: A 65-year-old male presented with a rare lipoma in the quadrigeminal cistern manifesting as left abducens nerve paresis which was effectively treated by total extirpation. The usual neurological symptoms of lipoma in the quadrigeminal cistern are intracranial hypertension and hydrocephalus which can be successfully treated by shunt operation. Direct surgery is indicated when the symptoms are ascribed to localized symptoms, but tumor removal should be partial if the superior cerebellar arteries are involved.

Congenital triventricular hydrocephalus associated with a small lipoma in the quadrigeminal plate cistern

Abstract: A case of neonatal congenital triventricular hydrocephalus due to a small intracranial lipoma in the quadrigeminal plate cistern is reported The patient was treated by a ventriculoperitoneal shunt The mechanism of obstructive hydrocephalus caused by the intracranial lipoma is discussed

Intracranial lipoma in a laboratory rat.

Abstract: reported in a wide variety of laboratory, domestic, and wild anima1s.I Stress as a result of weaning, overcrowding, poor sanitation, shipping, and corticosteroid administration often precedes outbreaks of Tyzzer’s d i~ease .~ Asymptomatic carriers are suspected oftransmitting the organism to susceptible individuals.’ Although natural interspecies transmission has not been demonstrated, environmental contamination with B. piliformis spores from rodent and rabbit feces has been suggested as a source of infection.* The proposed pathogenesis of Tyzzer’s disease involves an oral route of infection resulting in a primary enteric infection with local proliferation of the organism, especially within the jejunum, ileum, and cecum. Dissemination via the portal circulation results in infection of other tissues, especially the liver. Lymphatic invasion has been proposed as the source of intestinal smooth muscle infe~t ion.~ There is a distinct tropism for certain cell types, such as intestinal epithelium, hepatocytes, myocardium, and intestinal smooth muscle, although the mechanism of this cellular tropism is unknown. In vitro production of cytotoxins by B. piliformis has been demonstrated and is the likely cause of the necrotizing lesions.8 The marked lymphoid depletion observed in this puppy is compatible with lymphoid destruction resulting from a previous viral infection or improper development of the immune system. Similar lesions occur following infection with canine distemper virus or canine parvovirus-2 in dogs and combined immunodeficiency syndromes as seen in Arabian foals and severe combined immunodeficient mice. lo In this puppy, severe immunosuppression may have resulted in a more widely disseminated infection than is typically seen in canine Tyzzer’s disease, or the infection may have been due to a particularly virulent biovar. Lesions compatible with active canine distemper virus or canine parvovirus-2 infections were not evident. An antemortern fecal enzyme-linked immunosorbent assay for canine parvovirus-2 antigen was negative. Canine Tyzzer’s disease typically occurs in 5-7canine distemper virus infection; immunosuppression has been suspected in other

Lipoma do corpo caloso com extensão extracraniana através de falha óssea frontal

Abstract: Intracranial lipoma are usually localized in the corpus callosum and rarely extends to the subgaleal region. Only eight cases of lipoma of the corpus callosum with extracranial extension were reported in the literature. Seizures and mental retardation were the most common clinical findings in these patients. Data from the literature show that resection of intracranial lipoma has catastrophic results. The resection must be restricted to the extracranial portion. We report a child with lipoma of the corpus callosum with extracranial extension with no neurologic deficit submitted to resection of the extracranial extension.